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Cell Death and Differentiation|November 12, 2005
Loss of PKD1 and loss of Bcl-2 elicit polycystic kidney disease through distinct mechanismsP Hughes, M Robati, W Lu, et al.Oncogene|November 15, 2011
Bim must be able to engage all pro-survival Bcl-2 family members for efficient tumor suppressionD Mérino, A Strasser, P BouilletCell Death and Differentiation|January 14, 2012
Alternative splicing of Bim and Erk-mediated Bim(EL) phosphorylation are dispensable for hematopoietic homeostasis in vivoC Clybouw, D Merino, T Nebl, et al.Cell Death & Disease|October 13, 2012
Bcl-2 family member Bcl-G is not a proapoptotic proteinM Giam, T Okamoto, J D Mintern, et al.Developmental Cell|November 16, 2001
Degenerative disorders caused by Bcl-2 deficiency prevented by loss of its BH3-only antagonist BimP Bouillet, S Cory, L C Zhang, et al.Cell Death & Disease|June 8, 2013
Consequences of the combined loss of BOK and BAK or BOK and BAXF Ke, P Bouillet, T Kaufmann, et al.Oncogene|May 27, 2014
Functional antagonism between pro-apoptotic BIM and anti-apoptotic BCL-XL in MYC-induced lymphomagenesisA R D Delbridge, S Grabow, P Bouillet, et al.Cell Death & Disease|October 10, 2014
Impact of conditional deletion of the pro-apoptotic BCL-2 family member BIM in miceM J Herold, R Stuchbery, D Mérino, et al.Cell Death & Disease|August 24, 2012
Detection of Bcl-2 family member Bcl-G in mouse tissues using new monoclonal antibodiesM Giam, J D Mintern, G J P Rautureau, et al.Dermatology Research and Practice|January 21, 2016
Evaluation of Subcision for the Correction of the Prominent Nasolabial FoldsR M Robati, F Abdollahimajd, A M RobatiPageof 86