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Mutation Research|August 8, 1996
A CHO mutant, UV40, that is sensitive to diverse mutagens and represents a new complementation group of mitomycin C sensitivityD B Busch, M Z Zdzienicka, A T Natarajan, et al.
Carcinogenesis|August 6, 2005
Reduced XPC DNA repair gene mRNA levels in clinically normal parents of xeroderma pigmentosum patientsSikandar G Khan, Kyu-Seon Oh, Tala Shahlavi, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 4, 2018
Nur77 serves as a molecular brake of the metabolic switch during T cell activation to restrict autoimmunityMarie Liebmann, Stephanie Hucke, Kathrin Koch, et al.
EMBO Reports|September 13, 2021
Ronin governs the metabolic capacity of the embryonic lineage for post-implantation developmentKirill Salewskij, Theresa Gross-Thebing, Elizabeth Ing-Simmons, et al.
The Journal of Investigative Dermatology|June 13, 2002
Relationship of neurologic degeneration to genotype in three xeroderma pigmentosum group G patientsSteffen Emmert, Hanoch Slor, David B Busch, et al.
The Journal of Investigative Dermatology|March 28, 2008
Xeroderma pigmentosum-variant patients from America, Europe, and AsiaHiroki Inui, Kyu-Seon Oh, Carine Nadem, et al.
Bioorganic & Medicinal Chemistry Letters|June 19, 2014
Strategies for the modulation of phase II metabolism in a series of PKCε inhibitorsJeremy J Clemens, Timothy Coon, Brett B Busch, et al.
The EMBO Journal|November 18, 2022
LUBAC assembles a ubiquitin signaling platform at mitochondria for signal amplification and transport of NF-κB to the nucleusZhixiao Wu, Lena A Berlemann, Verian Bader, et al.
ACS Medicinal Chemistry Letters|December 21, 2016
Discovery of Highly Potent Liver X Receptor β AgonistsEllen K Kick, Brett B Busch, Richard Martin, et al.
Science Translational Medicine|May 3, 2019
Teriflunomide treatment for multiple sclerosis modulates T cell mitochondrial respiration with affinity-dependent effectsLuisa Klotz, Melanie Eschborn, Maren Lindner, et al.
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