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Clinical Infectious Diseases : an Official Publication of the Infectious Diseases Society of America|November 17, 2004
The influence of human N-acetyltransferase genotype on the early bactericidal activity of isoniazidP R Donald, F A Sirgel, A Venter, et al.Journal of Clinical Microbiology|November 28, 2002
Microevolution of the direct repeat region of Mycobacterium tuberculosis: implications for interpretation of spoligotyping dataR M Warren, E M Streicher, S L Sampson, et al.The Journal of Infection|July 9, 2002
Molecular analysis of clinical isolates of Mycobacterium tuberculosis collected from patients with persistent disease in the Khartoum region of SudanG S Sharaf-Eldin, N S Saeed, M E Hamid, et al.Journal of Clinical Microbiology|September 5, 2015
Molecular Epidemiological Interpretation of the Epidemic of Extensively Drug-Resistant Tuberculosis in South AfricaE M Streicher, S L Sampson, K Dheda, et al.Journal of Clinical Microbiology|February 15, 2008
Spread of a low-fitness drug-resistant Mycobacterium tuberculosis strain in a setting of high human immunodeficiency virus prevalenceO J Strauss, R M Warren, A Jordaan, et al.Journal of Clinical Microbiology|December 8, 2004
Clonal expansion of a globally disseminated lineage of Mycobacterium tuberculosis with low IS6110 copy numbersR M Warren, T C Victor, E M Streicher, et al.The International Journal of Tuberculosis and Lung Disease : the Official Journal of the International Union Against Tuberculosis and Lung Disease|December 16, 2006
An outbreak of drug-resistant tuberculosis caused by a Beijing strain in the western Cape, South AfricaR Johnson, R Warren, O J Strauss, et al.Clinical Infectious Diseases : an Official Publication of the Infectious Diseases Society of America|January 20, 2006
Bacille Calmette-Guérin vaccine-induced disease in HIV-infected and HIV-uninfected childrenA C Hesseling, H Rabie, B J Marais, et al.Journal of Clinical Microbiology|March 16, 2007
A recently evolved sublineage of the Mycobacterium tuberculosis Beijing strain family is associated with an increased ability to spread and cause diseaseM Hanekom, G D van der Spuy, E Streicher, et al.American Journal of Respiratory and Critical Care Medicine|May 1, 1997
Trimodality of isoniazid elimination: phenotype and genotype in patients with tuberculosisD P Parkin, S Vandenplas, F J Botha, et al.Pageof 15