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British Journal of Haematology|June 19, 2001
Complexes of anti-prothrombin antibodies and prothrombin cause lupus anticoagulant activity by competing with the binding of clotting factors for catalytic phospholipid surfacesM J Simmelink, D A Horbach, R H Derksen, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|May 1, 1996
Platelet adhesion to multimeric and dimeric von Willebrand factor and to collagen type III preincubated with von Willebrand factorY P Wu, H H van Breugel, H Lankhof, et al.
The Journal of Cardiovascular Surgery|June 5, 2004
In vivo transluminal microvascular endothelial cell seeding on balloon injured rabbit arteriesC H Arts, P G De Groot, N Attevelt, et al.
International Journal of Laboratory Hematology|January 17, 2018
High prevalence of reduced thrombin generation and/or decreased platelet response in women with unexplained heavy menstrual bleedingH P Eising, M Roest, P G de Groot, et al.
Journal of Thrombosis and Haemostasis : JTH|December 17, 2009
Mlck1a is expressed in zebrafish thrombocytes and is an essential component of thrombus formationE Tournoij, G J Weber, J W N Akkerman, et al.
Lupus|August 2, 2003
Catastrophic antiphospholipid syndrome: international consensus statement on classification criteria and treatment guidelinesR A Asherson, R Cervera, P G de Groot, et al.
American Journal of Obstetrics and Gynecology|July 1, 1992
A prospective, controlled multicenter study on the obstetric risks of pregnant women with antiphospholipid antibodiesH J Out, H W Bruinse, G C Christiaens, et al.
Thrombosis and Haemostasis|December 2, 1991
In vitro studies of antiphospholipid antibodies and its cofactor, beta 2-glycoprotein I, show negligible effects on endothelial cell mediated protein C activationJ D Oosting, R H Derksen, T M Hackeng, et al.
Arteriosclerosis and Thrombosis : a Journal of Vascular Biology|August 1, 1993
Aggregate formation is more strongly inhibited at high shear rates by dRGDW, a synthetic RGD-containing peptideE U Saelman, K M Hese, H K Nieuwenhuis, et al.
Annals of the New York Academy of Sciences|July 20, 2001
Mutations on fibrinogen (gamma 316-322) are associated with reduction in platelet adhesion under flow conditionsJ A Remijn, K C Lounes, K A Hogan, et al.
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