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Amyotrophic Lateral Sclerosis & Frontotemporal Degeneration|February 19, 2021
Better understanding the neurobiology of primary lateral sclerosisP Hande Ozdinler, Mukesh Gautam, Oge Gozutok, et al.
Clinical and Translational Medicine|February 26, 2021
Improving mitochondria and ER stability helps eliminate upper motor neuron degeneration that occurs due to mSOD1 toxicity and TDP-43 pathologyBarış Genç, Mukesh Gautam, Öge Gözütok, et al.
Frontiers in Molecular Neuroscience|June 9, 2020
The Electrophysiological Determinants of Corticospinal Motor Neuron Vulnerability in ALSJavier H Jara, Patrick L Sheets, Maximiliano José Nigro, et al.
Mutation Research|December 1, 1996
Mechanisms of induction of chromosomal aberrations and their detection by fluorescence in situ hybridizationA T Natarajan, A S Balajee, J J Boei, et al.
Scientific Reports|January 3, 2025
High-density multielectrode arrays bring cellular resolution to neuronal activity and network analyses of corticospinal motor neuronsChristopher A Quintanilla, Zachary Fitzgerald, Omar Kashow, et al.
Plos One|July 30, 2015
Visualization of Sensory Neurons and Their Projections in an Upper Motor Neuron Reporter LineBarış Genç, Amiko Krisa Bunag Lagrimas, Pınar Kuru, et al.
Neurobiology of Disease|June 19, 2026
Proteomic analysis reveals early pathological defects in corticospinal motor neurons of a spastin model of hereditary spastic paraplegia, which are improved by NU-9 treatmentMukesh Gautam, Mercedes Priego, Christopher Quintanilla, et al.
Scientific Reports|February 7, 2017
Apical dendrite degeneration, a novel cellular pathology for Betz cells in ALSBarış Genç, Javier H Jara, Amiko K B Lagrimas, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 3, 2013
eGFP expression under UCHL1 promoter genetically labels corticospinal motor neurons and a subpopulation of degeneration-resistant spinal motor neurons in an ALS mouse modelMarina V Yasvoina, Baris Genç, Javier H Jara, et al.
Genes & Development|November 23, 2000
Ku acts in a unique way at the mammalian telomere to prevent end joiningH L Hsu, D Gilley, S A Galande, et al.
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