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Showing results (11-20 of 17) with videos related to

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Clinical and Translational Medicine|February 26, 2021
Improving mitochondria and ER stability helps eliminate upper motor neuron degeneration that occurs due to mSOD1 toxicity and TDP-43 pathologyBarış Genç, Mukesh Gautam, Öge Gözütok, et al.
Cerebral Cortex (New York, N.Y. : 1991)|January 18, 2015
Corticospinal Motor Neurons Are Susceptible to Increased ER Stress and Display Profound Degeneration in the Absence of UCHL1 FunctionJavier H Jara, Barış Genç, Gregory A Cox, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 3, 2013
eGFP expression under UCHL1 promoter genetically labels corticospinal motor neurons and a subpopulation of degeneration-resistant spinal motor neurons in an ALS mouse modelMarina V Yasvoina, Baris Genç, Javier H Jara, et al.
Scientific Reports|February 7, 2017
Apical dendrite degeneration, a novel cellular pathology for Betz cells in ALSBarış Genç, Javier H Jara, Amiko K B Lagrimas, et al.
Journal of Neuroinflammation|June 28, 2017
Evidence for an early innate immune response in the motor cortex of ALSJavier H Jara, Barış Genç, Macdonell J Stanford, et al.
Annals of Clinical and Translational Neurology|May 28, 2016
Absence of UCHL 1 function leads to selective motor neuropathyBarış Genç, Javier H Jara, Megan C Schultz, et al.
Neurobiology of Disease|January 30, 2023
SBT-272 improves TDP-43 pathology in ALS upper motor neurons by modulating mitochondrial integrity, motility, and functionMukesh Gautam, Barış Genç, Benjamin Helmold, et al.
Pageof 2

Showing results (11-20 of 17) with videos related to

Sort By:
Pageof 2
You have reached the last page of results.This site can display upto 17 results.
Clinical and Translational Medicine|February 26, 2021
Improving mitochondria and ER stability helps eliminate upper motor neuron degeneration that occurs due to mSOD1 toxicity and TDP-43 pathologyBarış Genç, Mukesh Gautam, Öge Gözütok, et al.
Cerebral Cortex (New York, N.Y. : 1991)|January 18, 2015
Corticospinal Motor Neurons Are Susceptible to Increased ER Stress and Display Profound Degeneration in the Absence of UCHL1 FunctionJavier H Jara, Barış Genç, Gregory A Cox, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 3, 2013
eGFP expression under UCHL1 promoter genetically labels corticospinal motor neurons and a subpopulation of degeneration-resistant spinal motor neurons in an ALS mouse modelMarina V Yasvoina, Baris Genç, Javier H Jara, et al.
Scientific Reports|February 7, 2017
Apical dendrite degeneration, a novel cellular pathology for Betz cells in ALSBarış Genç, Javier H Jara, Amiko K B Lagrimas, et al.
Journal of Neuroinflammation|June 28, 2017
Evidence for an early innate immune response in the motor cortex of ALSJavier H Jara, Barış Genç, Macdonell J Stanford, et al.
Annals of Clinical and Translational Neurology|May 28, 2016
Absence of UCHL 1 function leads to selective motor neuropathyBarış Genç, Javier H Jara, Megan C Schultz, et al.
Neurobiology of Disease|January 30, 2023
SBT-272 improves TDP-43 pathology in ALS upper motor neurons by modulating mitochondrial integrity, motility, and functionMukesh Gautam, Barış Genç, Benjamin Helmold, et al.
Pageof 2