Search research articles
Contact Us
Filters
Showing results (251-260 of 343) with videos related to
Page
of 35
Sort By:
American Journal of Kidney Diseases : the Official Journal of the National Kidney Foundation
|
July 1, 1997
Vascular access survival among incident hemodialysis patients in the United States
J D Woods, M N Turenne, R L Strawderman, et al.
The Journal of Biological Chemistry
|
November 25, 1992
Interaction between the p21ras GTPase activating protein and the insulin receptor
G J Pronk, R H Medema, B M Burgering, et al.
Science (New York, N.Y.)
|
July 13, 1990
Inhibition of GTPase activating protein stimulation of Ras-p21 GTPase by the Krev-1 gene product
M Frech, J John, V Pizon, et al.
Nature Medicine
|
June 1, 1997
ONYX-015, an E1B gene-attenuated adenovirus, causes tumor-specific cytolysis and antitumoral efficacy that can be augmented by standard chemotherapeutic agents
C Heise, A Sampson-Johannes, A Williams, et al.
Science (New York, N.Y.)
|
October 4, 1985
A model for the tertiary structure of p21, the product of the ras oncogene
F McCormick, B F Clark, T F la Cour, et al.
American Journal of Kidney Diseases : the Official Journal of the National Kidney Foundation
|
July 21, 1998
Noncompliance in hemodialysis: predictors and survival analysis
J E Leggat, S M Orzol, T E Hulbert-Shearon, et al.
Cureus
|
January 27, 2025
Knee Ligament Injuries in the National Football League: Impact of Field Surface Type During the 2020-2023 Seasons
William F McCormick, Mitchell J Lomis, Christian B Cooper, et al.
Cancer Research
|
December 1, 1986
Detection of activated Mr 21,000 protein, the product of ras oncogenes, using antibodies with specificity for amino acid 12
G Wong, N Arnheim, R Clark, et al.
Science (New York, N.Y.)
|
January 10, 1992
GAP domains responsible for ras p21-dependent inhibition of muscarinic atrial K+ channel currents
G A Martin, A Yatani, R Clark, et al.
The New England Journal of Medicine
|
March 3, 1994
Loss of the normal NF1 allele from the bone marrow of children with type 1 neurofibromatosis and malignant myeloid disorders
K M Shannon, P O'Connell, G A Martin, et al.
Page
of 35
Search research articles
Search
Showing results (251-260 of 343) with videos related to
Sort By:
Page
of 35
American Journal of Kidney Diseases : the Official Journal of the National Kidney Foundation
|
July 1, 1997
Vascular access survival among incident hemodialysis patients in the United States
J D Woods, M N Turenne, R L Strawderman, et al.
The Journal of Biological Chemistry
|
November 25, 1992
Interaction between the p21ras GTPase activating protein and the insulin receptor
G J Pronk, R H Medema, B M Burgering, et al.
Science (New York, N.Y.)
|
July 13, 1990
Inhibition of GTPase activating protein stimulation of Ras-p21 GTPase by the Krev-1 gene product
M Frech, J John, V Pizon, et al.
Nature Medicine
|
June 1, 1997
ONYX-015, an E1B gene-attenuated adenovirus, causes tumor-specific cytolysis and antitumoral efficacy that can be augmented by standard chemotherapeutic agents
C Heise, A Sampson-Johannes, A Williams, et al.
Science (New York, N.Y.)
|
October 4, 1985
A model for the tertiary structure of p21, the product of the ras oncogene
F McCormick, B F Clark, T F la Cour, et al.
American Journal of Kidney Diseases : the Official Journal of the National Kidney Foundation
|
July 21, 1998
Noncompliance in hemodialysis: predictors and survival analysis
J E Leggat, S M Orzol, T E Hulbert-Shearon, et al.
Cureus
|
January 27, 2025
Knee Ligament Injuries in the National Football League: Impact of Field Surface Type During the 2020-2023 Seasons
William F McCormick, Mitchell J Lomis, Christian B Cooper, et al.
Cancer Research
|
December 1, 1986
Detection of activated Mr 21,000 protein, the product of ras oncogenes, using antibodies with specificity for amino acid 12
G Wong, N Arnheim, R Clark, et al.
Science (New York, N.Y.)
|
January 10, 1992
GAP domains responsible for ras p21-dependent inhibition of muscarinic atrial K+ channel currents
G A Martin, A Yatani, R Clark, et al.
The New England Journal of Medicine
|
March 3, 1994
Loss of the normal NF1 allele from the bone marrow of children with type 1 neurofibromatosis and malignant myeloid disorders
K M Shannon, P O'Connell, G A Martin, et al.
Page
of 35