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Experimental Parasitology|January 1, 1990
Plasmodium vivax: malarial proteins associated with the membrane-bound caveola-vesicle complexes and cytoplasmic cleft structures of infected erythrocytesJ W Barnwell, P Ingravallo, M R Galinski, et al.
Immunology Letters|August 1, 1990
Human cerebral malaria in Thailand: a clinico-pathological correlationM Riganti, E Pongponratn, T Tegoshi, et al.
Transplantation Proceedings|August 1, 1989
Functions of vascular wall cells related to development of transplantation-associated coronary arteriosclerosisP Libby, R N Salomon, D D Payne, et al.
Arteriosclerosis and Thrombosis : a Journal of Vascular Biology|November 1, 1994
The omega-3 fatty acid docosahexaenoate reduces cytokine-induced expression of proatherogenic and proinflammatory proteins in human endothelial cellsR De Caterina, M I Cybulsky, S K Clinton, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|August 1, 1996
Circumferential stress and matrix metalloproteinase 1 in human coronary atherosclerosis. Implications for plaque ruptureR T Lee, F J Schoen, H M Loree, et al.
Circulation|February 24, 2001
PPARalpha activators inhibit tissue factor expression and activity in human monocytesN Marx, N Mackman, U Schönbeck, et al.
Journal of Immunology (Baltimore, Md. : 1950)|October 1, 1989
Activated endothelial cells resist lymphokine-activated killer cell-mediated injury. Possible role of induced cytokines in limiting capillary leak during IL-2 therapyJ W Mier, E P Brandon, P Libby, et al.
Cell|April 7, 1989
PDGF-dependent tyrosine phosphorylation stimulates production of novel polyphosphoinositides in intact cellsK R Auger, L A Serunian, S P Soltoff, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 22, 1999
Cutting edge: heat shock protein (HSP) 60 activates the innate immune response: CD14 is an essential receptor for HSP60 activation of mononuclear cellsA Kol, A H Lichtman, R W Finberg, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 1, 1988
IL-2 rapidly induces natural killer cell adhesion to human endothelial cells. A potential mechanism for endothelial injuryF R Aronson, P Libby, E P Brandon, et al.
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