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Journal of Leukocyte Biology|February 19, 1998
Insulin-stimulated chemokinesis in normal human neutrophils is dependent on D-glucose concentration and sensitive to inhibitors of tyrosine kinase and phosphatidylinositol 3-kinaseP A Oldenborg, J SehlinBioscience Reports|June 7, 2000
The glucose concentration modulates N-formyl-methionyl-leucyl-phenylalanine (fMet-Leu-Phe)-stimulated chemokinesis in normal human neutrophilsP A Oldenborg, J SehlinThe American Journal of Physiology|November 1, 1988
Furosemide reduces insulin release by inhibition of Cl- and Ca2+ fluxes in beta-cellsP E Sandström, J SehlinEuropean Journal of Pharmacology|March 15, 1988
Furosemide-induced glucose intolerance in mice is associated with reduced insulin secretionP E Sandström, J SehlinBioscience Reports|January 7, 1998
Effects of D-glucose on chemokinesis and resting production of reactive oxygen species in neutrophil granulocytes of lean or obese-hyperglycemic mouseP A Oldenborg, J SehlinActa Physiologica Scandinavica|January 1, 1988
Furosemide causes acute and long-term hyperglycaemia and reduces glucose tolerance in miceP E Sandström, J SehlinBritish Journal of Pharmacology|June 1, 1981
Further studies on 5-hydroxytryptamine transport in pancreatic islets and isolated beta-cellsP LindströmActa Endocrinologica|June 1, 1984
Kinetics of 5-hydroxytryptophan potentiation of glucose-induced insulin releaseP LindströmBiochimica Et Biophysica Acta|November 19, 1986
Aromatic-L-amino-acid decarboxylase activity in mouse pancreatic isletsP LindströmEuropean Journal of Pharmacology|August 30, 2001
Modulation of islet ATP content by inhibition or stimulation of the Na(+)/K(+) pumpA Elmi, L Idahl, J SehlinPageof 20