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Cells|June 27, 2020
Deferiprone (DFP) Targets Cancer Stem Cell (CSC) Propagation by Inhibiting Mitochondrial Metabolism and Inducing ROS ProductionMarco Fiorillo, Fanni Tóth, Matteo Brindisi, et al.
Frontiers in Oncology|August 16, 2021
MitoTracker Deep Red (MTDR) Is a Metabolic Inhibitor for Targeting Mitochondria and Eradicating Cancer Stem Cells (CSCs), With Anti-Tumor and Anti-Metastatic Activity In VivoCamillo Sargiacomo, Sophie Stonehouse, Zahra Moftakhar, et al.
The Journal of Biological Chemistry|August 28, 1999
Phenotypic behavior of caveolin-3 mutations that cause autosomal dominant limb girdle muscular dystrophy (LGMD-1C). Retention of LGMD-1C caveolin-3 mutants within the golgi complexF Galbiati, D Volonte, C Minetti, et al.
Science (New York, N.Y.)|September 23, 1988
Insulin-stimulated release of lipoprotein lipase by metabolism of its phosphatidylinositol anchorB L Chan, M P Lisanti, E Rodriguez-Boulan, et al.
The Journal of Biological Chemistry|September 7, 2000
Limb-girdle muscular dystrophy (LGMD-1C) mutants of caveolin-3 undergo ubiquitination and proteasomal degradation. Treatment with proteasomal inhibitors blocks the dominant negative effect of LGMD-1C mutanta and rescues wild-type caveolin-3F Galbiati, D Volonte, C Minetti, et al.
Neurology|February 26, 2004
Caveolinopathies: mutations in caveolin-3 cause four distinct autosomal dominant muscle diseasesS E Woodman, F Sotgia, F Galbiati, et al.
Cell Metabolism|December 5, 2015
Metastasis and Oxidative Stress: Are Antioxidants a Metabolic Driver of Progression?Maria Peiris-Pagès, Ubaldo E Martinez-Outschoorn, Federica Sotgia, et al.
The Journal of Membrane Biology|February 1, 1990
Preferred apical distribution of glycosyl-phosphatidylinositol (GPI) anchored proteins: a highly conserved feature of the polarized epithelial cell phenotypeM P Lisanti, A Le Bivic, A R Saltiel, et al.
Oncotarget|April 16, 2017
Mitochondrial "power" drives tamoxifen resistance: NQO1 and GCLC are new therapeutic targets in breast cancerMarco Fiorillo, Federica Sotgia, Diego Sisci, et al.
American Journal of Physiology. Heart and Circulatory Physiology|January 18, 2006
Caveolin-1-deficient aortic smooth muscle cells show cell autonomous abnormalities in proliferation, migration, and endothelin-based signal transductionGhada S Hassan, Terence M Williams, Philippe G Frank, et al.
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