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Proceedings of the National Academy of Sciences of the United States of America|October 1, 1990
Vectorial apical delivery and slow endocytosis of a glycolipid-anchored fusion protein in transfected MDCK cellsM P Lisanti, I W Caras, T Gilbert, et al.Cell Cycle (Georgetown, Tex.)|May 15, 2007
Localized treatment with a novel FDA-approved proteasome inhibitor blocks the degradation of dystrophin and dystrophin-associated proteins in mdx miceGloria Bonuccelli, Federica Sotgia, Franco Capozza, et al.Oncotarget|November 8, 2017
Mitoketoscins: Novel mitochondrial inhibitors for targeting ketone metabolism in cancer stem cells (CSCs)Bela Ozsvari, Federica Sotgia, Katie Simmons, et al.Circulation|August 31, 2006
Short-term administration of a cell-permeable caveolin-1 peptide prevents the development of monocrotaline-induced pulmonary hypertension and right ventricular hypertrophyJean-François Jasmin, Isabelle Mercier, Jocelyn Dupuis, et al.Giornale Italiano Di Cardiologia|October 1, 1987
[Idiopathic dilatation of the atria: a syndrome? Description of a case of idiopathic biatrial dilatation, review of the literature and a proposal for a pathogenetic interpretation]F Accorsi, G Caruso, R Fiorilli, et al.Giornale Italiano Di Cardiologia|November 1, 1990
[Coronary angioplasty in patients with compromised left ventricular function]A Colombo, P Lisanti, W Serino, et al.FEBS Letters|December 9, 1998
Upregulation of caveolin-1 and caveolae organelles in Taxol-resistant A549 cellsC P Yang, F Galbiati, D Volonte, et al.Cell Cycle (Georgetown, Tex.)|December 17, 2005
Mitochondrial impairment is a critical event in anthrax lethal toxin-induced cytolysis of murine macrophagesAbdelkrim Alileche, Raynal C Squires, Stefan M Muehlbauer, et al.The Journal of Biological Chemistry|April 23, 1999
Flotillins/cavatellins are differentially expressed in cells and tissues and form a hetero-oligomeric complex with caveolins in vivo. Characterization and epitope-mapping of a novel flotillin-1 monoclonal antibody probeD Volonte, F Galbiati, S Li, et al.The Journal of Biological Chemistry|October 9, 1999
Targeted down-regulation of caveolin-3 is sufficient to inhibit myotube formation in differentiating C2C12 myoblasts. Transient activation of p38 mitogen-activated protein kinase is required for induction of caveolin-3 expression and subsequent myotube formationF Galbiati, D Volonte, J A Engelman, et al.Pageof 58