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Immunotherapy|February 25, 2017
Anti-CTLA-4 therapy for malignant mesotheliomaAlice Guazzelli, Emyr Bakker, Marija Krstic-Demonacos, et al.Aging|July 8, 2020
Essential role of STAT5a in DCIS formation and invasion following estrogen treatmentSundee Dees, Laura Pontiggia, Jean-Francois Jasmin, et al.The Journal of Biological Chemistry|January 24, 1997
Identification, sequence, and expression of an invertebrate caveolin gene family from the nematode Caenorhabditis elegans. Implications for the molecular evolution of mammalian caveolin genesZ Tang, T Okamoto, P Boontrakulpoontawee, et al.The American Journal of Pathology|December 1, 2012
Caveolin-1 deficiency induces spontaneous endothelial-to-mesenchymal transition in murine pulmonary endothelial cells in vitroZhaodong Li, Peter J Wermuth, Bryan S Benn, et al.Oncogene|September 1, 1994
In vitro phosphorylation of caveolin-rich membrane domains: identification of an associated serine kinase activity as a casein kinase II-like enzymeM Sargiacomo, P E Scherer, Z L Tang, et al.The Journal of Biological Chemistry|November 5, 1999
p42/44 MAP kinase-dependent and -independent signaling pathways regulate caveolin-1 gene expression. Activation of Ras-MAP kinase and protein kinase a signaling cascades transcriptionally down-regulates caveolin-1 promoter activityJ A Engelman, X L Zhang, B Razani, et al.Biochemistry|November 29, 2001
Tyrosine phosphorylation of beta-dystroglycan at its WW domain binding motif, PPxY, recruits SH2 domain containing proteinsF Sotgia, H Lee, M T Bedford, et al.American Journal of Physiology. Cell Physiology|May 30, 2008
Role of caveolin-1 in the regulation of lipoprotein metabolismPhilippe G Frank, Stephanos Pavlides, Michelle W-C Cheung, et al.Cancer Research|November 17, 2006
Caveolin-1, mammary stem cells, and estrogen-dependent breast cancersFederica Sotgia, Hallgeir Rui, Gloria Bonuccelli, et al.Oncotarget|November 22, 2014
Mitochondria as new therapeutic targets for eradicating cancer stem cells: Quantitative proteomics and functional validation via MCT1/2 inhibitionRebecca Lamb, Hannah Harrison, James Hulit, et al.Pageof 58