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Cell Cycle (Georgetown, Tex.)|January 12, 2012
Is cancer a metabolic rebellion against host aging? In the quest for immortality, tumor cells try to save themselves by boosting mitochondrial metabolismAdam Ertel, Aristotelis Tsirigos, Diana Whitaker-Menezes, et al.
American Journal of Physiology. Renal Physiology|November 13, 2009
Postobstructive regeneration of kidney is derailed when surge in renal stem cells during course of unilateral ureteral obstruction is haltedH C Park, K Yasuda, B Ratliff, et al.
Cancer Research|November 20, 2015
Cyclin D1 Promotes Androgen-Dependent DNA Damage Repair in Prostate Cancer CellsMathew C Casimiro, Gabriele Di Sante, Xiaoming Ju, et al.
Cancer Research|August 17, 2010
CAV1 inhibits metastatic potential in melanomas through suppression of the integrin/Src/FAK signaling pathwayCasey Trimmer, Diana Whitaker-Menezes, Gloria Bonuccelli, et al.
Cancer Biology & Therapy|September 24, 2010
Understanding the "lethal" drivers of tumor-stroma co-evolution: emerging role(s) for hypoxia, oxidative stress and autophagy/mitophagy in the tumor micro-environmentMichael P Lisanti, Ubaldo E Martinez-Outschoorn, Barbara Chiavarina, et al.
Neurology|April 4, 2000
Mutation in the CAV3 gene causes partial caveolin-3 deficiency and hyperCKemiaI Carbone, C Bruno, F Sotgia, et al.
The American Journal of Pathology|February 20, 2010
Transcription factor Stat3 stimulates metastatic behavior of human prostate cancer cells in vivo, whereas Stat5b has a preferential role in the promotion of prostate cancer cell viability and tumor growthLei Gu, Ayush Dagvadorj, Jacqueline Lutz, et al.
Cancer Research|March 8, 2012
Genetic ablation of Cav1 differentially affects melanoma tumor growth and metastasis in mice: role of Cav1 in Shh heterotypic signaling and transendothelial migrationFranco Capozza, Casey Trimmer, Remedios Castello-Cros, et al.
Molecular Biology of the Cell|October 22, 2002
Caveolin-1-deficient mice show accelerated mammary gland development during pregnancy, premature lactation, and hyperactivation of the Jak-2/STAT5a signaling cascadeDavid S Park, Hyangkyu Lee, Philippe G Frank, et al.
Cancer Biology & Therapy|November 2, 2011
Anti-estrogen resistance in breast cancer is induced by the tumor microenvironment and can be overcome by inhibiting mitochondrial function in epithelial cancer cellsUbaldo E Martinez-Outschoorn, Allison Goldberg, Zhao Lin, et al.
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