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Structure (London, England : 1993)|December 24, 1998
The crystal structure of pneumococcal surface antigen PsaA reveals a metal-binding site and a novel structure for a putative ABC-type binding proteinM C Lawrence, P A Pilling, V C Epa, et al.The Journal of Infectious Diseases|January 15, 2008
A pneumococcal MerR-like regulator and S-nitrosoglutathione reductase are required for systemic virulenceUwe H Stroeher, Stephen P Kidd, Sian L Stafford, et al.The Journal of Infectious Diseases|September 1, 1995
The limited role of pneumolysin in the pathogenesis of pneumococcal meningitisI R Friedland, M M Paris, S Hickey, et al.Infection and Immunity|September 24, 2004
The ClpP protease of Streptococcus pneumoniae modulates virulence gene expression and protects against fatal pneumococcal challengeHyog-Young Kwon, A David Ogunniyi, Moo-Hyun Choi, et al.Infection and Immunity|September 30, 2015
Isolation site influences virulence phenotype of serotype 14 Streptococcus pneumoniae strains belonging to multilocus sequence type 15Zarina Amin, Richard M Harvey, Hui Wang, et al.Traffic (Copenhagen, Denmark)|August 15, 2009
The COG complex, Rab6 and COPI define a novel Golgi retrograde trafficking pathway that is exploited by SubAB toxinRichard D Smith, Rose Willett, Tetyana Kudlyk, et al.Antimicrobial Agents and Chemotherapy|June 10, 2010
Evaluation of biophotonic imaging to estimate bacterial burden in mice infected with highly virulent compared to less virulent Streptococcus pneumoniae serotypesStefanie Henken, Jennifer Bohling, A David Ogunniyi, et al.Journal of Analytical Toxicology|March 9, 2013
False-positive buprenorphine by CEDIA in patients prescribed amisulpride or sulpirideM A Birch, L Couchman, S Pietromartire, et al.The Journal of Biological Chemistry|August 2, 2012
The Escherichia coli subtilase cytotoxin A subunit specifically cleaves cell-surface GRP78 protein and abolishes COOH-terminal-dependent signalingRupa Ray, Gustaaf G de Ridder, Jerry P Eu, et al.Human Genetics|September 10, 1999
A common PEX1 frameshift mutation in patients with disorders of peroxisome biogenesis correlates with the severe Zellweger syndrome phenotypeM A Maxwell, P V Nelson, S J Chin, et al.Pageof 57