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Nature Genetics|February 2, 2000
LMNA, encoding lamin A/C, is mutated in partial lipodystrophyS Shackleton, D J Lloyd, S N Jackson, et al.Diabetologia|October 31, 2008
Apolipoproteins, cardiovascular risk and statin response in type 2 diabetes: the Collaborative Atorvastatin Diabetes Study (CARDS)V Charlton-Menys, D J Betteridge, H Colhoun, et al.The Cochrane Database of Systematic Reviews|October 21, 2004
Omega 3 fatty acids for prevention and treatment of cardiovascular diseaseL Hooper, R L Thompson, R A Harrison, et al.Current Molecular Medicine|June 14, 2011
The role of combined assessment of defense against oxidative stress and inflammation in the evaluation of peripheral arterial diseaseA Rull, R García, L Fernández-Sender, et al.Biochemical and Biophysical Research Communications|March 1, 2000
Paraoxonase activity is reduced by a pro-atherosclerotic diet in rabbitsM Mackness, A Boullier, N Hennuyer, et al.Journal of Medical Genetics|December 5, 2006
Genetic causes of familial hypercholesterolaemia in patients in the UK: relation to plasma lipid levels and coronary heart disease riskS E Humphries, R A Whittall, C S Hubbart, et al.Diabetic Medicine : a Journal of the British Diabetic Association|December 21, 2010
Protection Against Nephropathy in Diabetes with Atorvastatin (PANDA): a randomized double-blind placebo-controlled trial of high- vs. low-dose atorvastatin(1)M K Rutter, H R Prais, V Charlton-Menys, et al.Journal of Internal Medicine|February 16, 2006
Apo B versus cholesterol in estimating cardiovascular risk and in guiding therapy: report of the thirty-person/ten-country panelP J Barter, C M Ballantyne, R Carmena, et al.Pageof 17