Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

P P Dzeja

Showing results (1-10 of 18) with videos related to

Pageof 2
Sort By:
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|May 12, 1998
Phosphotransfer reactions in the regulation of ATP-sensitive K+ channelsP P Dzeja, A Terzic
Journal of Molecular and Cellular Cardiology|October 21, 2000
Mitochondrial K(ATP) channels: probing molecular identity and pharmacologyA Terzic, P P Dzeja, E L Holmuhamedov
Molecular and Cellular Biochemistry|September 24, 1998
Adenylate kinase: kinetic behavior in intact cells indicates it is integral to multiple cellular processesP P Dzeja, R J Zeleznikar, N D Goldberg
The Journal of Biological Chemistry|May 31, 1996
Suppression of creatine kinase-catalyzed phosphotransfer results in increased phosphoryl transfer by adenylate kinase in intact skeletal muscleP P Dzeja, R J Zeleznikar, N D Goldberg
The Journal of Biological Chemistry|March 31, 1995
Adenylate kinase-catalyzed phosphoryl transfer couples ATP utilization with its generation by glycolysis in intact muscleR J Zeleznikar, P P Dzeja, N D Goldberg
Current Cardiology Reports|September 12, 2000
Failing energetics in failing heartsP P Dzeja, M M Redfield, J C Burnett, et al.
The American Journal of Physiology|November 14, 1998
Mitochondrial ATP-sensitive K+ channels modulate cardiac mitochondrial functionE L Holmuhamedov, S Jovanović, P P Dzeja, et al.
The Journal of Biological Chemistry|October 5, 2001
Cellular energetics in the preconditioned state: protective role for phosphotransfer reactions captured by 18O-assisted 31P NMRD Pucar, P P Dzeja, P Bast, et al.
Molecular and Cellular Biochemistry|January 12, 2000
Reduced activity of enzymes coupling ATP-generating with ATP-consuming processes in the failing myocardiumP P Dzeja, D Pucar, M M Redfield, et al.
Circulation Research|May 29, 1999
Adenylate kinase-catalyzed phosphotransfer in the myocardium : increased contribution in heart failureP P Dzeja, K T Vitkevicius, M M Redfield, et al.
Pageof 2

Showing results (1-10 of 18) with videos related to

Sort By:
Pageof 2
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|May 12, 1998
Phosphotransfer reactions in the regulation of ATP-sensitive K+ channelsP P Dzeja, A Terzic
Journal of Molecular and Cellular Cardiology|October 21, 2000
Mitochondrial K(ATP) channels: probing molecular identity and pharmacologyA Terzic, P P Dzeja, E L Holmuhamedov
Molecular and Cellular Biochemistry|September 24, 1998
Adenylate kinase: kinetic behavior in intact cells indicates it is integral to multiple cellular processesP P Dzeja, R J Zeleznikar, N D Goldberg
The Journal of Biological Chemistry|May 31, 1996
Suppression of creatine kinase-catalyzed phosphotransfer results in increased phosphoryl transfer by adenylate kinase in intact skeletal muscleP P Dzeja, R J Zeleznikar, N D Goldberg
The Journal of Biological Chemistry|March 31, 1995
Adenylate kinase-catalyzed phosphoryl transfer couples ATP utilization with its generation by glycolysis in intact muscleR J Zeleznikar, P P Dzeja, N D Goldberg
Current Cardiology Reports|September 12, 2000
Failing energetics in failing heartsP P Dzeja, M M Redfield, J C Burnett, et al.
The American Journal of Physiology|November 14, 1998
Mitochondrial ATP-sensitive K+ channels modulate cardiac mitochondrial functionE L Holmuhamedov, S Jovanović, P P Dzeja, et al.
The Journal of Biological Chemistry|October 5, 2001
Cellular energetics in the preconditioned state: protective role for phosphotransfer reactions captured by 18O-assisted 31P NMRD Pucar, P P Dzeja, P Bast, et al.
Molecular and Cellular Biochemistry|January 12, 2000
Reduced activity of enzymes coupling ATP-generating with ATP-consuming processes in the failing myocardiumP P Dzeja, D Pucar, M M Redfield, et al.
Circulation Research|May 29, 1999
Adenylate kinase-catalyzed phosphotransfer in the myocardium : increased contribution in heart failureP P Dzeja, K T Vitkevicius, M M Redfield, et al.
Pageof 2