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P Piccardo

Showing results (51-60 of 58) with videos related to

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Nature Genetics|November 14, 1997
Lack of apolipoprotein E dramatically reduces amyloid beta-peptide depositionK R Bales, T Verina, R C Dodel, et al.
The Journal of Biological Chemistry|November 23, 2000
A 7-kDa prion protein (PrP) fragment, an integral component of the PrP region required for infectivity, is the major amyloid protein in Gerstmann-Sträussler-Scheinker disease A117VF Tagliavini, P M Lievens, C Tranchant, et al.
Acta Neuropathologica|July 27, 2000
Senile dementia associated with amyloid beta protein angiopathy and tau perivascular pathology but not neuritic plaques in patients homozygous for the APOE-epsilon4 alleleR Vidal, M Calero, P Piccardo, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 23, 1996
Vascular variant of prion protein cerebral amyloidosis with tau-positive neurofibrillary tangles: the phenotype of the stop codon 145 mutation in PRNPB Ghetti, P Piccardo, M G Spillantini, et al.
Annals of Neurology|August 12, 1999
Classification of sporadic Creutzfeldt-Jakob disease based on molecular and phenotypic analysis of 300 subjectsP Parchi, A Giese, S Capellari, et al.
The American Journal of Pathology|June 8, 2001
Prion proteins with different conformations accumulate in Gerstmann-Sträussler-Scheinker disease caused by A117V and F198S mutationsP Piccardo, J J Liepnieks, A William, et al.
Neurology|February 13, 2002
Insomnia associated with thalamic involvement in E200K Creutzfeldt-Jakob diseaseAna Lia Taratuto, P Piccardo, E G Reich, et al.
Journal of Neuropathology and Experimental Neurology|October 24, 1998
Phenotypic variability of Gerstmann-Sträussler-Scheinker disease is associated with prion protein heterogeneityP Piccardo, S R Dlouhy, P M Lievens, et al.
Pageof 6

Showing results (51-60 of 58) with videos related to

Sort By:
Pageof 6
You have reached the last page of results.This site can display upto 58 results.
Nature Genetics|November 14, 1997
Lack of apolipoprotein E dramatically reduces amyloid beta-peptide depositionK R Bales, T Verina, R C Dodel, et al.
The Journal of Biological Chemistry|November 23, 2000
A 7-kDa prion protein (PrP) fragment, an integral component of the PrP region required for infectivity, is the major amyloid protein in Gerstmann-Sträussler-Scheinker disease A117VF Tagliavini, P M Lievens, C Tranchant, et al.
Acta Neuropathologica|July 27, 2000
Senile dementia associated with amyloid beta protein angiopathy and tau perivascular pathology but not neuritic plaques in patients homozygous for the APOE-epsilon4 alleleR Vidal, M Calero, P Piccardo, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 23, 1996
Vascular variant of prion protein cerebral amyloidosis with tau-positive neurofibrillary tangles: the phenotype of the stop codon 145 mutation in PRNPB Ghetti, P Piccardo, M G Spillantini, et al.
Annals of Neurology|August 12, 1999
Classification of sporadic Creutzfeldt-Jakob disease based on molecular and phenotypic analysis of 300 subjectsP Parchi, A Giese, S Capellari, et al.
The American Journal of Pathology|June 8, 2001
Prion proteins with different conformations accumulate in Gerstmann-Sträussler-Scheinker disease caused by A117V and F198S mutationsP Piccardo, J J Liepnieks, A William, et al.
Neurology|February 13, 2002
Insomnia associated with thalamic involvement in E200K Creutzfeldt-Jakob diseaseAna Lia Taratuto, P Piccardo, E G Reich, et al.
Journal of Neuropathology and Experimental Neurology|October 24, 1998
Phenotypic variability of Gerstmann-Sträussler-Scheinker disease is associated with prion protein heterogeneityP Piccardo, S R Dlouhy, P M Lievens, et al.
Pageof 6