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P Poorkaj

Showing results (1-10 of 32) with videos related to

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Current Psychiatry Reports|December 21, 2000
The genetics of Alzheimer's diseaseG D Schellenberg, I D'Souza, P Poorkaj
Genomics|August 22, 2000
Single-step conversion of P1 and P1 artificial chromosome clones into yeast artificial chromosomesP Poorkaj, K R Peterson, G D Schellenberg
Archives of Neurology|November 16, 2001
Impact of DNA testing for early-onset familial Alzheimer disease and frontotemporal dementiaE J Steinbart, C O Smith, P Poorkaj, et al.
Hepatology (Baltimore, Md.)|September 1, 1995
Organellar clusters formed by mitochondrial-rough endoplasmic reticulum associations: an ordered arrangement of mitochondria in hepatocytesJ Cascarano, P A Chambers, E Schwartz, et al.
The Journal of Histochemistry and Cytochemistry : Official Journal of the Histochemistry Society|November 1, 1996
Neuronal expression of STM2 mRNA in human brain is reduced in Alzheimer's diseaseP J McMillan, J B Leverenz, P Poorkaj, et al.
Mammalian Genome : Official Journal of the International Mammalian Genome Society|October 20, 2001
A genomic sequence analysis of the mouse and human microtubule-associated protein tauP Poorkaj, A Kas, I D'Souza, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 13, 1999
Missense and silent tau gene mutations cause frontotemporal dementia with parkinsonism-chromosome 17 type, by affecting multiple alternative RNA splicing regulatory elementsI D'Souza, P Poorkaj, M Hong, et al.
Genomics|June 1, 1996
Genomic structure and expression of STM2, the chromosome 1 familial Alzheimer disease geneE Levy-Lahad, P Poorkaj, K Wang, et al.
American Journal of Medical Genetics. Part A|July 22, 2004
parkin mutation analysis in clinic patients with early-onset Parkinson [corrected] diseaseP Poorkaj, J G Nutt, D James, et al.
Neurology|February 26, 2000
Familial frontotemporal dementia with ubiquitin-positive, tau-negative inclusionsA Kertesz, T Kawarai, E Rogaeva, et al.
Pageof 4

Showing results (1-10 of 32) with videos related to

Sort By:
Pageof 4
Current Psychiatry Reports|December 21, 2000
The genetics of Alzheimer's diseaseG D Schellenberg, I D'Souza, P Poorkaj
Genomics|August 22, 2000
Single-step conversion of P1 and P1 artificial chromosome clones into yeast artificial chromosomesP Poorkaj, K R Peterson, G D Schellenberg
Archives of Neurology|November 16, 2001
Impact of DNA testing for early-onset familial Alzheimer disease and frontotemporal dementiaE J Steinbart, C O Smith, P Poorkaj, et al.
Hepatology (Baltimore, Md.)|September 1, 1995
Organellar clusters formed by mitochondrial-rough endoplasmic reticulum associations: an ordered arrangement of mitochondria in hepatocytesJ Cascarano, P A Chambers, E Schwartz, et al.
The Journal of Histochemistry and Cytochemistry : Official Journal of the Histochemistry Society|November 1, 1996
Neuronal expression of STM2 mRNA in human brain is reduced in Alzheimer's diseaseP J McMillan, J B Leverenz, P Poorkaj, et al.
Mammalian Genome : Official Journal of the International Mammalian Genome Society|October 20, 2001
A genomic sequence analysis of the mouse and human microtubule-associated protein tauP Poorkaj, A Kas, I D'Souza, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 13, 1999
Missense and silent tau gene mutations cause frontotemporal dementia with parkinsonism-chromosome 17 type, by affecting multiple alternative RNA splicing regulatory elementsI D'Souza, P Poorkaj, M Hong, et al.
Genomics|June 1, 1996
Genomic structure and expression of STM2, the chromosome 1 familial Alzheimer disease geneE Levy-Lahad, P Poorkaj, K Wang, et al.
American Journal of Medical Genetics. Part A|July 22, 2004
parkin mutation analysis in clinic patients with early-onset Parkinson [corrected] diseaseP Poorkaj, J G Nutt, D James, et al.
Neurology|February 26, 2000
Familial frontotemporal dementia with ubiquitin-positive, tau-negative inclusionsA Kertesz, T Kawarai, E Rogaeva, et al.
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