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P R Shepherd

Showing results (31-40 of 58) with videos related to

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Biological Trace Element Research|January 1, 1992
Glucose tolerance factor potentiation of insulin action in adipocytes from rats raised on a torula yeast diet cannot be attributed to a deficiency of chromium or glucose tolerance factor activity in the dietP R Shepherd, C Elwood, P D Buckley, et al.
FEBS Letters|November 11, 1999
The alpha-isoform of class II phosphoinositide 3-kinase is more effectively activated by insulin receptors than IGF receptors, and activation requires receptor NPEY motifsB Ursø, R A Brown, S O'Rahilly, et al.
The Biochemical Journal|August 19, 2000
Comparison of the kinetic properties of the lipid- and protein-kinase activities of the p110alpha and p110beta catalytic subunits of class-Ia phosphoinositide 3-kinasesC A Beeton, E M Chance, L C Foukas, et al.
Biochemical Society Transactions|August 28, 2002
Conditionally immortalized cell lines as model systems for high-throughput biology in drug discoveryN Daniele, R Halse, E Grinyo, et al.
Diabetologia|October 10, 2008
Atypical antipsychotic drugs induce derangements in glucose homeostasis by acutely increasing glucagon secretion and hepatic glucose output in the ratG C Smith, C Chaussade, M Vickers, et al.
The Journal of Biological Chemistry|May 18, 1999
Insulin activates the alpha isoform of class II phosphoinositide 3-kinaseR A Brown, J Domin, A Arcaro, et al.
Endocrinology|March 1, 1995
High level overexpression of glucose transporter-4 driven by an adipose-specific promoter is maintained in transgenic mice on a high fat diet, but does not prevent impaired glucose toleranceL Gnudi, E Tozzo, P R Shepherd, et al.
The Biochemical Journal|November 24, 1999
Mammalian target of rapamycin is a direct target for protein kinase B: identification of a convergence point for opposing effects of insulin and amino-acid deficiency on protein translationB T Navé, M Ouwens, D J Withers, et al.
The Biochemical Journal|August 15, 1996
Compartment-specific regulation of phosphoinositide 3-kinase by platelet-derived growth factor and insulin in 3T3-L1 adipocytesB T Navé, R J Haigh, A C Hayward, et al.
The Journal of Biological Chemistry|October 25, 1993
Adipose cell hyperplasia and enhanced glucose disposal in transgenic mice overexpressing GLUT4 selectively in adipose tissueP R Shepherd, L Gnudi, E Tozzo, et al.
Pageof 6

Showing results (31-40 of 58) with videos related to

Sort By:
Pageof 6
Biological Trace Element Research|January 1, 1992
Glucose tolerance factor potentiation of insulin action in adipocytes from rats raised on a torula yeast diet cannot be attributed to a deficiency of chromium or glucose tolerance factor activity in the dietP R Shepherd, C Elwood, P D Buckley, et al.
FEBS Letters|November 11, 1999
The alpha-isoform of class II phosphoinositide 3-kinase is more effectively activated by insulin receptors than IGF receptors, and activation requires receptor NPEY motifsB Ursø, R A Brown, S O'Rahilly, et al.
The Biochemical Journal|August 19, 2000
Comparison of the kinetic properties of the lipid- and protein-kinase activities of the p110alpha and p110beta catalytic subunits of class-Ia phosphoinositide 3-kinasesC A Beeton, E M Chance, L C Foukas, et al.
Biochemical Society Transactions|August 28, 2002
Conditionally immortalized cell lines as model systems for high-throughput biology in drug discoveryN Daniele, R Halse, E Grinyo, et al.
Diabetologia|October 10, 2008
Atypical antipsychotic drugs induce derangements in glucose homeostasis by acutely increasing glucagon secretion and hepatic glucose output in the ratG C Smith, C Chaussade, M Vickers, et al.
The Journal of Biological Chemistry|May 18, 1999
Insulin activates the alpha isoform of class II phosphoinositide 3-kinaseR A Brown, J Domin, A Arcaro, et al.
Endocrinology|March 1, 1995
High level overexpression of glucose transporter-4 driven by an adipose-specific promoter is maintained in transgenic mice on a high fat diet, but does not prevent impaired glucose toleranceL Gnudi, E Tozzo, P R Shepherd, et al.
The Biochemical Journal|November 24, 1999
Mammalian target of rapamycin is a direct target for protein kinase B: identification of a convergence point for opposing effects of insulin and amino-acid deficiency on protein translationB T Navé, M Ouwens, D J Withers, et al.
The Biochemical Journal|August 15, 1996
Compartment-specific regulation of phosphoinositide 3-kinase by platelet-derived growth factor and insulin in 3T3-L1 adipocytesB T Navé, R J Haigh, A C Hayward, et al.
The Journal of Biological Chemistry|October 25, 1993
Adipose cell hyperplasia and enhanced glucose disposal in transgenic mice overexpressing GLUT4 selectively in adipose tissueP R Shepherd, L Gnudi, E Tozzo, et al.
Pageof 6