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Journal of Medical Genetics|March 4, 2005
Association of oestrogen receptor alpha gene polymorphisms with postmenopausal bone loss, bone mass, and quantitative ultrasound properties of boneO M E Albagha, U Pettersson, A Stewart, et al.
Calcified Tissue International|December 1, 1992
Factors predicting the acute effect of pamidronate on serum calcium in hypercalcemia of malignancyS J Gallacher, W D Fraser, F C Logue, et al.
Postgraduate Medical Journal|November 1, 1990
Immobilization-related hypercalcaemia--a possible novel mechanism and response to pamidronateS J Gallacher, S H Ralston, F J Dryburgh, et al.
Family Practice|February 7, 2007
Non-cardiac chest pain: a retrospective cohort study of patients who attended a Rapid Access Chest Pain ClinicJ C Dumville, H MacPherson, K Griffith, et al.
Arthritis and Rheumatism|January 5, 2002
Activation of the inducible nitric oxide synthase pathway contributes to inflammation-induced osteoporosis by suppressing bone formation and causing osteoblast apoptosisK J Armour, K E Armour, R J van't Hof, et al.
The Journal of Clinical Investigation|April 4, 2001
A COL1A1 Sp1 binding site polymorphism predisposes to osteoporotic fracture by affecting bone density and qualityV Mann, E E Hobson, B Li, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 28, 2000
Requirement of the inducible nitric oxide synthase pathway for IL-1-induced osteoclastic bone resorptionR J van't Hof, K J Armour, L M Smith, et al.
Gait & Posture|June 2, 2020
Laboratory-Based Gait Variability and Habitual Gait Entropy Do Not Differentiate Community-Dwelling Older Adults from Those with Subjective Memory ComplaintsT Rantalainen, W P Teo, N D Ridgers, et al.
Calcified Tissue International|May 13, 2006
Loss of ubiquitin binding is a unifying mechanism by which mutations of SQSTM1 cause Paget's disease of boneJ R Cavey, S H Ralston, P W Sheppard, et al.
Diabetologia|January 20, 2004
Association of the Pro12Ala and C1431T variants of PPARG and their haplotypes with susceptibility to Type 2 diabetesA S F Doney, B Fischer, J E Cecil, et al.
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