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Showing results (51-60 of 58) with videos related to

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Genomics|April 1, 1997
Identification of a new human catenin gene family member (ARVCF) from the region deleted in velo-cardio-facial syndromeH Sirotkin, H O'Donnell, R DasGupta, et al.
The American Journal of Psychiatry|February 1, 1996
No evidence for allelic association between schizophrenia and a polymorphism determining high or low catechol O-methyltransferase activityJ K Daniels, N M Williams, J Williams, et al.
American Journal of Human Genetics|June 12, 1999
Mutations of UFD1L are not responsible for the majority of cases of DiGeorge Syndrome/velocardiofacial syndrome without deletions within chromosome 22q11R Wadey, J McKie, C Papapetrou, et al.
American Journal of Human Genetics|October 27, 1997
Molecular definition of 22q11 deletions in 151 velo-cardio-facial syndrome patientsC Carlson, H Sirotkin, R Pandita, et al.
American Journal of Human Genetics|April 1, 1996
Human homologue sequences to the Drosophila dishevelled segment-polarity gene are deleted in the DiGeorge syndromeA Pizzuti, G Novelli, A Mari, et al.
Nature Genetics|December 8, 1998
A homeobox gene, HLXB9, is the major locus for dominantly inherited sacral agenesisA J Ross, V Ruiz-Perez, Y Wang, et al.
Cell|March 10, 2001
TBX1 is responsible for cardiovascular defects in velo-cardio-facial/DiGeorge syndromeS Merscher, B Funke, J A Epstein, et al.
American Journal of Human Genetics|April 6, 2000
Mutation analysis and embryonic expression of the HLXB9 Currarino syndrome geneD M Hagan, A J Ross, T Strachan, et al.
Pageof 6

Showing results (51-60 of 58) with videos related to

Sort By:
Pageof 6
You have reached the last page of results.This site can display upto 58 results.
Genomics|April 1, 1997
Identification of a new human catenin gene family member (ARVCF) from the region deleted in velo-cardio-facial syndromeH Sirotkin, H O'Donnell, R DasGupta, et al.
The American Journal of Psychiatry|February 1, 1996
No evidence for allelic association between schizophrenia and a polymorphism determining high or low catechol O-methyltransferase activityJ K Daniels, N M Williams, J Williams, et al.
American Journal of Human Genetics|June 12, 1999
Mutations of UFD1L are not responsible for the majority of cases of DiGeorge Syndrome/velocardiofacial syndrome without deletions within chromosome 22q11R Wadey, J McKie, C Papapetrou, et al.
American Journal of Human Genetics|October 27, 1997
Molecular definition of 22q11 deletions in 151 velo-cardio-facial syndrome patientsC Carlson, H Sirotkin, R Pandita, et al.
American Journal of Human Genetics|April 1, 1996
Human homologue sequences to the Drosophila dishevelled segment-polarity gene are deleted in the DiGeorge syndromeA Pizzuti, G Novelli, A Mari, et al.
Nature Genetics|December 8, 1998
A homeobox gene, HLXB9, is the major locus for dominantly inherited sacral agenesisA J Ross, V Ruiz-Perez, Y Wang, et al.
Cell|March 10, 2001
TBX1 is responsible for cardiovascular defects in velo-cardio-facial/DiGeorge syndromeS Merscher, B Funke, J A Epstein, et al.
American Journal of Human Genetics|April 6, 2000
Mutation analysis and embryonic expression of the HLXB9 Currarino syndrome geneD M Hagan, A J Ross, T Strachan, et al.
Pageof 6