Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

P Seubert

Showing results (41-50 of 53) with videos related to

Pageof 6
Sort By:
Annals of the New York Academy of Sciences|February 24, 2001
Prevention and reduction of AD-type pathology in PDAPP mice immunized with A beta 1-42D Games, F Bard, H Grajeda, et al.
Neuroscience Letters|October 21, 1998
Cerebrospinal fluid tau protein is not elevated in HIV-associated neurologic disease in humans. HIV Neurobehavioral Research Center Group (HNRC)R J Ellis, P Seubert, R Motter, et al.
The Journal of Biological Chemistry|June 5, 1990
The protease inhibitory properties of the Alzheimer's beta-amyloid precursor proteinS Sinha, H F Dovey, P Seubert, et al.
Vaccine|July 18, 2001
Encapsulation in biodegradable microparticles enhances serum antibody response to parenterally-delivered beta-amyloid in miceD J Brayden, L Templeton, S McClean, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 18, 1997
Amyloid precursor protein processing and A beta42 deposition in a transgenic mouse model of Alzheimer diseaseK Johnson-Wood, M Lee, R Motter, et al.
Archives of Neurology|July 25, 1998
High cerebrospinal fluid tau and low amyloid beta42 levels in the clinical diagnosis of Alzheimer disease and relation to apolipoprotein E genotypeD Galasko, L Chang, R Motter, et al.
Neurobiology of Disease|January 1, 1997
Presenilin proteins undergo heterogeneous endoproteolysis between Thr291 and Ala299 and occur as stable N- and C-terminal fragments in normal and Alzheimer brain tissueM B Podlisny, M Citron, P Amarante, et al.
Annals of Neurology|June 1, 1997
A novel presenilin-1 mutation: increased beta-amyloid and neurofibrillary changesT Gómez-Isla, W Wasco, W P Pettingell, et al.
Neurobiology of Aging|January 4, 2011
Amyloid precursor protein (APP) processing genes and cerebrospinal fluid APP cleavage product levels in Alzheimer's diseaseL M Bekris, N M Galloway, S Millard, et al.
Nature Medicine|August 10, 2000
Peripherally administered antibodies against amyloid beta-peptide enter the central nervous system and reduce pathology in a mouse model of Alzheimer diseaseF Bard, C Cannon, R Barbour, et al.
Pageof 6

Showing results (41-50 of 53) with videos related to

Sort By:
Pageof 6
Annals of the New York Academy of Sciences|February 24, 2001
Prevention and reduction of AD-type pathology in PDAPP mice immunized with A beta 1-42D Games, F Bard, H Grajeda, et al.
Neuroscience Letters|October 21, 1998
Cerebrospinal fluid tau protein is not elevated in HIV-associated neurologic disease in humans. HIV Neurobehavioral Research Center Group (HNRC)R J Ellis, P Seubert, R Motter, et al.
The Journal of Biological Chemistry|June 5, 1990
The protease inhibitory properties of the Alzheimer's beta-amyloid precursor proteinS Sinha, H F Dovey, P Seubert, et al.
Vaccine|July 18, 2001
Encapsulation in biodegradable microparticles enhances serum antibody response to parenterally-delivered beta-amyloid in miceD J Brayden, L Templeton, S McClean, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 18, 1997
Amyloid precursor protein processing and A beta42 deposition in a transgenic mouse model of Alzheimer diseaseK Johnson-Wood, M Lee, R Motter, et al.
Archives of Neurology|July 25, 1998
High cerebrospinal fluid tau and low amyloid beta42 levels in the clinical diagnosis of Alzheimer disease and relation to apolipoprotein E genotypeD Galasko, L Chang, R Motter, et al.
Neurobiology of Disease|January 1, 1997
Presenilin proteins undergo heterogeneous endoproteolysis between Thr291 and Ala299 and occur as stable N- and C-terminal fragments in normal and Alzheimer brain tissueM B Podlisny, M Citron, P Amarante, et al.
Annals of Neurology|June 1, 1997
A novel presenilin-1 mutation: increased beta-amyloid and neurofibrillary changesT Gómez-Isla, W Wasco, W P Pettingell, et al.
Neurobiology of Aging|January 4, 2011
Amyloid precursor protein (APP) processing genes and cerebrospinal fluid APP cleavage product levels in Alzheimer's diseaseL M Bekris, N M Galloway, S Millard, et al.
Nature Medicine|August 10, 2000
Peripherally administered antibodies against amyloid beta-peptide enter the central nervous system and reduce pathology in a mouse model of Alzheimer diseaseF Bard, C Cannon, R Barbour, et al.
Pageof 6