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The American Journal of Physiology|November 1, 1995
Endothelium-dependent relaxation is depressed at the macro- and microcirculatory levels during sepsisP Wang, Z F Ba, I H ChaudryInternational Journal of Molecular Medicine|April 14, 2000
Mechanism of hepatocellular dysfunction during sepsis: the role of gut-derived norepinephrine (review)D J Koo, I H Chaudry, P WangThe Journal of Surgical Research|November 1, 1996
Pentoxifylline restores the depressed cardiac performance after trauma-hemorrhage and resuscitationD A Robinson, P Wang, I H ChaudryThe American Journal of Physiology|April 1, 1995
ATP-MgCl2 restores depressed endothelial cell function after hemorrhagic shock and resuscitationP Wang, Z F Ba, I H ChaudryArchives of Surgery (Chicago, Ill. : 1960)|February 1, 1991
Hepatic extraction of indocyanine green is depressed early in sepsis despite increased hepatic blood flow and cardiac outputP Wang, Z F Ba, I H ChaudryBiochimica Et Biophysica Acta|November 21, 1997
Administration of ATP-MgCl2 following hemorrhage and resuscitation increases hepatic phosphoenolpyruvate carboxykinase and decreases pyruvate kinase activitiesM S Mahmoud, P Wang, I H ChaudryThe American Journal of Physiology|December 1, 1992
Differential effects of ATP-MgCl2 on portal and hepatic arterial blood flow after hemorrhage and resuscitationP Wang, Z F Ba, I H ChaudryArchives of Surgery (Chicago, Ill. : 1960)|April 1, 1997
Mechanism of hepatocellular dysfunction during early sepsis. Key role of increased gene expression and release of proinflammatory cytokines tumor necrosis factor and interleukin-6P Wang, Z F Ba, I H ChaudryBiochimica Et Biophysica Acta|June 6, 2000
The dissociation between upregulated endothelins and hemodynamic responses during polymicrobial sepsisD A Ornan, I H Chaudry, P WangThe American Journal of Physiology|June 1, 1994
Administration of tumor necrosis factor-alpha in vivo depresses endothelium-dependent relaxationP Wang, Z F Ba, I H ChaudryPageof 497