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Archives of Surgery (Chicago, Ill. : 1960)|November 1, 1994
Nitric oxide. To block or enhance its production during sepsis?P Wang, Z F Ba, I H ChaudryCirculatory Shock|April 1, 1992
ATP-MgCl2 restores the depressed cardiac output following trauma and severe hemorrhage even in the absence of blood resuscitationP Wang, Z F Ba, I H ChaudryShock (Augusta, Ga.)|January 1, 1995
Hepatocellular dysfunction occurs earlier than the onset of hyperdynamic circulation during sepsisP Wang, Z F Ba, I H ChaudryThe American Journal of Physiology|December 1, 1991
Increase in hepatic blood flow during early sepsis is due to increased portal blood flowP Wang, Z F Ba, I H ChaudryThe Journal of Trauma|March 1, 1997
Pentoxifylline maintains hepatocellular function and improves cardiac performance during early sepsisP Wang, Z F Ba, I H ChaudryShock (Augusta, Ga.)|July 1, 1996
A novel nonanticoagulant heparin prevents vascular endothelial cell dysfunction during hyperdynamic sepsisA M Morrison, P Wang, I H ChaudryThe Journal of Surgical Research|February 15, 1997
Liver endothelial cell function is depressed only during hypodynamic sepsisP Wang, Z F Ba, I H ChaudryCirculatory Shock|December 1, 1990
Hemorrhage produces depression in microvascular blood flow which persists despite fluid resuscitationP Wang, J G Hauptman, I H ChaudrySurgery|August 1, 1994
Chemically modified heparin improves hepatocellular function, cardiac output, and microcirculation after trauma-hemorrhage and resuscitationP Wang, Z F Ba, I H ChaudryBiochimica Et Biophysica Acta|July 10, 1997
Severe hypoxemia in the absence of blood loss depresses hepatocellular function and up-regulates IL-6 and PGE2P Wang, Z F Ba, I H ChaudryPageof 497