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HGG Advances|July 7, 2025
Haploinsufficient variants in SMAD5 are associated with isolated congenital heart diseaseDimuthu Alankarage, Iryna Leshchynska, Stephanie Portelli, et al.
Cell|November 22, 2025
Inhibition of oligomeric BAX by an anti-apoptotic dimerCatherine E Newman, Micah A Gygi, Haleh Alimohamadi, et al.
Science Advances|October 4, 2024
The generation of stable microvessels in ischemia is mediated by endothelial cell derived TRAILSiân P Cartland, Manisha S Patil, Elaina Kelland, et al.
Scientific Reports|September 11, 2023
Fibulin-3 is necessary to prevent cardiac rupture following myocardial infarctionLucy A Murtha, Sean A Hardy, Nishani S Mabotuwana, et al.
Nature Communications|January 30, 2021
Single cell sequencing reveals endothelial plasticity with transient mesenchymal activation after myocardial infarctionLukas S Tombor, David John, Simone F Glaser, et al.
Cell|March 14, 2007
An Nkx2-5/Bmp2/Smad1 negative feedback loop controls heart progenitor specification and proliferationOwen W J Prall, Mary K Menon, Mark J Solloway, et al.
Science (New York, N.Y.)|September 12, 2024
Solvent-mediated oxide hydrogenation in layered cathodesGang Wan, Travis P Pollard, Lin Ma, et al.
JCI Insight|March 30, 2017
Point mutations in murine <i>Nkx2-5</i> phenocopy human congenital heart disease and induce pathogenic Wnt signalingMilena B Furtado, Julia C Wilmanns, Anjana Chandran, et al.
The Journal of Cell Biology|June 22, 2011
Tinman/Nkx2-5 acts via miR-1 and upstream of Cdc42 to regulate heart function across speciesLi Qian, Joshua D Wythe, Jiandong Liu, et al.
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