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Cancer Cell|April 18, 2006
Ras ubiquitination: coupling spatial sorting and signal transmissionPablo Rodriguez-Viciana, Frank McCormickMethods in Enzymology|June 8, 2006
Characterization of interactions between ras family GTPases and their effectorsPablo Rodriguez-Viciana, Frank McCormickMolecular and Cellular Biology|May 15, 2004
Signaling specificity by Ras family GTPases is determined by the full spectrum of effectors they regulatePablo Rodriguez-Viciana, Celine Sabatier, Frank McCormickMolecular Cell|April 25, 2006
A phosphatase holoenzyme comprised of Shoc2/Sur8 and the catalytic subunit of PP1 functions as an M-Ras effector to modulate Raf activityPablo Rodriguez-Viciana, Juan Oses-Prieto, Alma Burlingame, et al.Cancer Cell|August 16, 2005
A conditional feedback loop regulates Ras activity through EphA2Madhu Macrae, Richard M Neve, Pablo Rodriguez-Viciana, et al.Methods in Enzymology|April 17, 2008
Biochemical characterization of novel germline BRAF and MEK mutations in cardio-facio-cutaneous syndromePablo Rodriguez-Viciana, Katherine A RauenCold Spring Harbor Perspectives in Medicine|January 10, 2018
MRAS: A Close but Understudied Member of the RAS FamilyLucy C Young, Pablo Rodriguez-VicianaProceedings of the National Academy of Sciences of the United States of America|December 13, 2006
Polyoma and SV40 proteins differentially regulate PP2A to activate distinct cellular signaling pathways involved in growth controlPablo Rodriguez-Viciana, Crista Collins, Mike FriedScience (New York, N.Y.)|January 28, 2006
Germline mutations in genes within the MAPK pathway cause cardio-facio-cutaneous syndromePablo Rodriguez-Viciana, Osamu Tetsu, William E Tidyman, et al.Pageof 30