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Nature Communications|November 25, 2018
Mediated nuclear import and export of TAZ and the underlying molecular requirementsMichael Kofler, Pam Speight, Darby Little, et al.
Molecular Biology of the Cell|January 10, 2014
The fate of the primary cilium during myofibroblast transitionMatthew Rozycki, Monika Lodyga, Jessica Lam, et al.
Molecular Biology of the Cell|February 8, 2013
Central role of the exchange factor GEF-H1 in TNF-α-induced sequential activation of Rac, ADAM17/TACE, and RhoA in tubular epithelial cellsFaiza Waheed, Qinghong Dan, Yasaman Amoozadeh, et al.
American Journal of Physiology. Cell Physiology|December 26, 2008
Hyperosmotic stress induces Rho/Rho kinase/LIM kinase-mediated cofilin phosphorylation in tubular cells: key role in the osmotically triggered F-actin responseAna C P Thirone, Pam Speight, Matthew Zulys, et al.
The Journal of Biological Chemistry|July 26, 2017
TGF-β1 regulates the expression and transcriptional activity of TAZ protein via a Smad3-independent, myocardin-related transcription factor-mediated mechanismMaria Zena Miranda, Janne Folke Bialik, Pam Speight, et al.
FEBS Letters|December 25, 2007
Rac, PAK and p38 regulate cell contact-dependent nuclear translocation of myocardin-related transcription factorAttila Sebe, András Masszi, Matthew Zulys, et al.
The Journal of Biological Chemistry|March 6, 2009
GEF-H1 mediates tumor necrosis factor-alpha-induced Rho activation and myosin phosphorylation: role in the regulation of tubular paracellular permeabilityEli Kakiashvili, Pam Speight, Faiza Waheed, et al.
Nature Materials|November 1, 2016
MicroRNA-21 preserves the fibrotic mechanical memory of mesenchymal stem cellsChen Xi Li, Nilesh P Talele, Stellar Boo, et al.
American Journal of Physiology. Cell Physiology|October 12, 2012
Hyperosmotic stress regulates the distribution and stability of myocardin-related transcription factor, a key modulator of the cytoskeletonDonald L Ly, Faiza Waheed, Monika Lodyga, et al.
The Journal of Biological Chemistry|September 5, 2019
Claudin-2 suppresses GEF-H1, RHOA, and MRTF, thereby impacting proliferation and profibrotic phenotype of tubular cellsQinghong Dan, Yixuan Shi, Razieh Rabani, et al.
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