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BMC Medical Genomics|November 1, 2014
A common gene expression signature in Huntington's disease patient brain regionsAndreas Neueder, Gillian P BatesThe Journal of Clinical Investigation|February 3, 2011
The importance of integrating basic and clinical research toward the development of new therapies for Huntington diseaseIgnacio Munoz-Sanjuan, Gillian P BatesMethods in Molecular Biology (Clifton, N.J.)|June 18, 2004
Mouse models of triplet repeat diseasesGillian P Bates, David G HayMethods in Molecular Biology (Clifton, N.J.)|June 18, 2004
Monitoring aggregate formation in organotypic slice cultures from transgenic miceDonna L Smith, Gillian P BatesMolecular Neurodegeneration|October 29, 2008
Optimisation of region-specific reference gene selection and relative gene expression analysis methods for pre-clinical trials of Huntington's diseaseCaroline L Benn, Helen Fox, Gillian P BatesPlos One|July 3, 2015
In Vivo Profiling Reveals a Competent Heat Shock Response in Adult Neurons: Implications for Neurodegenerative DisordersAlisia Carnemolla, Hayley Lazell, Saliha Moussaoui, et al.Plos One|June 17, 2011
Hdac6 knock-out increases tubulin acetylation but does not modify disease progression in the R6/2 mouse model of Huntington's diseaseAnna Bobrowska, Paolo Paganetti, Patrick Matthias, et al.The Journal of Biological Chemistry|May 24, 2014
Novel isoforms of heat shock transcription factor 1, HSF1γα and HSF1γβ, regulate chaperone protein gene transcriptionAndreas Neueder, Francesca Achilli, Saliha Moussaoui, et al.Plos One|April 9, 2009
The ubiquitin-proteasome reporter GFPu does not accumulate in neurons of the R6/2 transgenic mouse model of Huntington's diseaseJohn S Bett, Casey Cook, Leonard Petrucelli, et al.Current Opinion in Neurology|November 11, 2005
Biomarkers for neurodegenerative diseasesSusie M D Henley, Gillian P Bates, Sarah J TabriziPageof 14