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Science Advances|June 15, 2022
GPR15L is an epithelial inflammation-derived pruritogenPang-Yen Tseng, Mark A HoonScience Translational Medicine|November 10, 2021
Oncostatin M can sensitize sensory neurons in inflammatory pruritusPang-Yen Tseng, Mark A HoonThe Journal of Investigative Dermatology|October 18, 2021
Interactions of the Neuro‒Immune‒Stromal Triad in ItchPang-Yen Tseng, Mark A HoonHandbook of Experimental Pharmacology|November 4, 2020
Molecular Genetics of Kappa Opioids in Pain and Itch SensationsPang-Yen Tseng, Mark A HoonThe Journal of Investigative Dermatology|September 4, 2021
Specific β-Defensins Stimulate Pruritus through Activation of Sensory NeuronsPang-Yen Tseng, Mark A HoonPain|July 24, 2025
Upregulated natriuretic peptide B expression as a hallmark of chronic itchPang-Yen Tseng, Huey Ling Liew, Matthias Ringkamp, et al.Cell Reports|March 28, 2019
Nppb Neurons Are Sensors of Mast Cell-Induced ItchHans Jürgen Solinski, Mette C Kriegbaum, Pang-Yen Tseng, et al.The Journal of General Physiology|August 1, 2007
Cytoplasmic ATP inhibition of CLC-1 is enhanced by low pHPang-Yen Tseng, Brett Bennetts, Tsung-Yu ChenItch (Philadelphia, Pa.)|January 14, 2021
MrgprX1 Mediates Neuronal Excitability and Itch Through Tetrodotoxin-Resistant Sodium ChannelsPang-Yen Tseng, Qin Zheng, Zhe Li, et al.The Journal of General Physiology|October 1, 2008
ATP inhibition of CLC-1 is controlled by oxidation and reductionXiao-Dong Zhang, Pang-Yen Tseng, Tsung-Yu ChenPageof 6