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Parisa Asghari

Showing results (11-20 of 21) with videos related to

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Protoplasma|November 8, 2011
The structure and functioning of the couplon in the mammalian cardiomyocyteParisa Asghari, David R L Scriven, Jeremy Hoskins, et al.
Elife|January 10, 2020
Cardiac ryanodine receptor distribution is dynamic and changed by auxiliary proteins and post-translational modificationParisa Asghari, David Rl Scriven, Myles Ng, et al.
Biorxiv : the Preprint Server for Biology|June 9, 2023
PHOSPHORYLATION OF RyR2 SIMULTANEOUSLY EXPANDS THE DYAD AND REARRANGES THE TETRAMERSParisa Asghari, David R L Scriven, Saba Shahrasebi, et al.
Circulation Research|May 3, 2014
Nonuniform and variable arrangements of ryanodine receptors within mammalian ventricular couplonsParisa Asghari, David R L Scriven, Shubhayan Sanatani, et al.
Frontiers in Cardiovascular Medicine|September 5, 2022
Using human induced pluripotent stem cell-derived cardiomyocytes to understand the mechanisms driving cardiomyocyte maturationHoma Hamledari, Parisa Asghari, Farah Jayousi, et al.
The Journal of General Physiology|February 22, 2024
Phosphorylation of RyR2 simultaneously expands the dyad and rearranges the tetramersParisa Asghari, David R L Scriven, Saba Shahrasebi, et al.
Frontiers in Cell and Developmental Biology|February 2, 2024
hiPSC-derived cardiomyocytes as a model to study the role of small-conductance Ca<sup>2+</sup>-activated K<sup>+</sup> (SK) ion channel variants associated with atrial fibrillationHosna Babini, Verónica Jiménez-Sábado, Ekaterina Stogova, et al.
Cardiovascular Research|August 8, 2012
Cardiac ryanodine receptors control heart rate and rhythmicity in adult miceMichael J Bround, Parisa Asghari, Rich B Wambolt, et al.
Current Protocols|December 27, 2021
Using hiPSC-CMs to Examine Mechanisms of Catecholaminergic Polymorphic Ventricular TachycardiaAlia Arslanova, Sanam Shafaattalab, Kevin Ye, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 23, 2009
Ablation of triadin causes loss of cardiac Ca2+ release units, impaired excitation-contraction coupling, and cardiac arrhythmiasNagesh Chopra, Tao Yang, Parisa Asghari, et al.
Pageof 3

Showing results (11-20 of 21) with videos related to

Sort By:
Pageof 3
Protoplasma|November 8, 2011
The structure and functioning of the couplon in the mammalian cardiomyocyteParisa Asghari, David R L Scriven, Jeremy Hoskins, et al.
Elife|January 10, 2020
Cardiac ryanodine receptor distribution is dynamic and changed by auxiliary proteins and post-translational modificationParisa Asghari, David Rl Scriven, Myles Ng, et al.
Biorxiv : the Preprint Server for Biology|June 9, 2023
PHOSPHORYLATION OF RyR2 SIMULTANEOUSLY EXPANDS THE DYAD AND REARRANGES THE TETRAMERSParisa Asghari, David R L Scriven, Saba Shahrasebi, et al.
Circulation Research|May 3, 2014
Nonuniform and variable arrangements of ryanodine receptors within mammalian ventricular couplonsParisa Asghari, David R L Scriven, Shubhayan Sanatani, et al.
Frontiers in Cardiovascular Medicine|September 5, 2022
Using human induced pluripotent stem cell-derived cardiomyocytes to understand the mechanisms driving cardiomyocyte maturationHoma Hamledari, Parisa Asghari, Farah Jayousi, et al.
The Journal of General Physiology|February 22, 2024
Phosphorylation of RyR2 simultaneously expands the dyad and rearranges the tetramersParisa Asghari, David R L Scriven, Saba Shahrasebi, et al.
Frontiers in Cell and Developmental Biology|February 2, 2024
hiPSC-derived cardiomyocytes as a model to study the role of small-conductance Ca<sup>2+</sup>-activated K<sup>+</sup> (SK) ion channel variants associated with atrial fibrillationHosna Babini, Verónica Jiménez-Sábado, Ekaterina Stogova, et al.
Cardiovascular Research|August 8, 2012
Cardiac ryanodine receptors control heart rate and rhythmicity in adult miceMichael J Bround, Parisa Asghari, Rich B Wambolt, et al.
Current Protocols|December 27, 2021
Using hiPSC-CMs to Examine Mechanisms of Catecholaminergic Polymorphic Ventricular TachycardiaAlia Arslanova, Sanam Shafaattalab, Kevin Ye, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 23, 2009
Ablation of triadin causes loss of cardiac Ca2+ release units, impaired excitation-contraction coupling, and cardiac arrhythmiasNagesh Chopra, Tao Yang, Parisa Asghari, et al.
Pageof 3