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Genetics|May 18, 2007
Defective decapentaplegic signaling results in heart overgrowth and reduced cardiac output in DrosophilaAaron N Johnson, Lindsey A Burnett, Julia Sellin, et al.
Genetics|January 14, 2003
BEST: a novel computational approach for comparing gene expression patterns from early stages of Drosophila melanogaster developmentSudhir Kumar, Karthik Jayaraman, Sethuraman Panchanathan, et al.
Plos One|May 28, 2010
A data integration approach to mapping OCT4 gene regulatory networks operative in embryonic stem cells and embryonal carcinoma cellsMarc Jung, Hedi Peterson, Lukas Chavez, et al.
Developmental Biology|December 11, 2007
A combinatorial enhancer recognized by Mad, TCF and Brinker first activates then represses dpp expression in the posterior spiracles of DrosophilaNorma T Takaesu, Denis S Bulanin, Aaron N Johnson, et al.
Comptes Rendus Biologies|November 14, 2009
ENFIN--A European network for integrative systems biologyPascal Kahlem, Andrew Clegg, Florian Reisinger, et al.
G3 (Bethesda, Md.)|July 15, 2018
CORL Expression and Function in Insulin Producing Neurons Reversibly Influences Adult Longevity in DrosophilaNancy L Tran, Samuel L Goldsmith, Agapi Dimitriadou, et al.
G3 (Bethesda, Md.)|January 27, 2022
New resources for the Drosophila 4th chromosome: FRT101F enabled mitotic clones and Bloom syndrome helicase enabled meiotic recombinationSamuel L Goldsmith, MaryJane Shimell, Petra Tauscher, et al.
Bioinformatics (Oxford, England)|December 5, 2013
Automated annotation of developmental stages of Drosophila embryos in images containing spatial patterns of expressionLei Yuan, Cheng Pan, Shuiwang Ji, et al.
Genetics|November 28, 2007
Phylogenetic and genomewide analyses suggest a functional relationship between kayak, the Drosophila fos homolog, and fig, a predicted protein phosphatase 2c nested within a kayak intronStephanie G Hudson, Matthew J Garrett, Joseph W Carlson, et al.
G3 (Bethesda, Md.)|March 28, 2026
Genetic analyses enabled by the Fourth Chromosome Resource Projectreveal unexpected mutant phenotypes and suggest new disease modelsBonnie M Weasner, Michael J Stinchfield, Brandon P Weasner, et al.
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