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Overview Leadership Blog JoVE Help Center

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Publishing Process Editorial Board Scope & Policies Peer Review FAQ Submit

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Testimonials Subscriptions Access Resources Library Advisory Board FAQ

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Patricia Outeda

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Nature Communications|October 3, 2025

SFPQ-TFE3 reciprocally regulates mTORC1 and induces lineage plasticity in a mouse model of renal tumorigenesisKaushal Asrani, Adrianna Amaral, Juhyung Woo, et al.

Scientific Reports|February 11, 2018

A cleavage product of Polycystin-1 is a mitochondrial matrix protein that affects mitochondria morphology and function when heterologously expressedCheng-Chao Lin, Mahiro Kurashige, Yi Liu, et al.

Scientific Reports|August 12, 2017

NEDD4-family E3 ligase dysfunction due to PKHD1/Pkhd1 defects suggests a mechanistic model for ARPKD pathobiologyJun-Ya Kaimori, Cheng-Chao Lin, Patricia Outeda, et al.

Journal of the American Society of Nephrology : JASN|February 10, 2026

A HaloTag Knock-In Resource for In Vivo and In Vitro Analysis of Endogenous Polycystin-2 Localization, Turnover, and TransportZhang Li, Courtney J Haycraft, Mandy J Croyle, et al.

American Journal of Human Genetics|December 10, 2021

Monoallelic IFT140 pathogenic variants are an important cause of the autosomal dominant polycystic kidney-spectrum phenotypeSarah R Senum, Ying Sabrina M Li, Katherine A Benson, et al.

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Search research articles

Showing results (21-30 of 25) with videos related to

Sort By:

Pageof 3

You have reached the last page of results.This site can display upto 25 results.

Nature Communications|October 3, 2025

SFPQ-TFE3 reciprocally regulates mTORC1 and induces lineage plasticity in a mouse model of renal tumorigenesisKaushal Asrani, Adrianna Amaral, Juhyung Woo, et al.

Scientific Reports|February 11, 2018

A cleavage product of Polycystin-1 is a mitochondrial matrix protein that affects mitochondria morphology and function when heterologously expressedCheng-Chao Lin, Mahiro Kurashige, Yi Liu, et al.

Scientific Reports|August 12, 2017

NEDD4-family E3 ligase dysfunction due to PKHD1/Pkhd1 defects suggests a mechanistic model for ARPKD pathobiologyJun-Ya Kaimori, Cheng-Chao Lin, Patricia Outeda, et al.

Journal of the American Society of Nephrology : JASN|February 10, 2026

A HaloTag Knock-In Resource for In Vivo and In Vitro Analysis of Endogenous Polycystin-2 Localization, Turnover, and TransportZhang Li, Courtney J Haycraft, Mandy J Croyle, et al.

American Journal of Human Genetics|December 10, 2021

Monoallelic IFT140 pathogenic variants are an important cause of the autosomal dominant polycystic kidney-spectrum phenotypeSarah R Senum, Ying Sabrina M Li, Katherine A Benson, et al.

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