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Scientific Reports|June 13, 2018
AKT signalling selectively regulates PINK1 mitophagy in SHSY5Y cells and human iPSC-derived neuronsMarc P M Soutar, Liam Kempthorne, Shuichi Miyakawa, et al.
FEBS Open Bio|October 16, 2023
LRRK2 negatively regulates glucose tolerance via regulation of membrane translocation of GLUT4 in adipocytesFumitaka Kawakami, Motoki Imai, Yuki Isaka, et al.
Brain : a Journal of Neurology|September 8, 2022
Regulation of mitophagy by the NSL complex underlies genetic risk for Parkinson's disease at 16q11.2 and MAPT H1 lociMarc P M Soutar, Daniela Melandri, Benjamin O'Callaghan, et al.
Annals of Neurology|December 11, 2008
Mitochondrial function and morphology are impaired in parkin-mutant fibroblastsHeather Mortiboys, Kelly Jean Thomas, Werner J H Koopman, et al.
The Journal of Biological Chemistry|July 10, 2003
L166P mutant DJ-1, causative for recessive Parkinson's disease, is degraded through the ubiquitin-proteasome systemDavid W Miller, Rili Ahmad, Stephen Hague, et al.
The International Journal of Neuropsychopharmacology|December 21, 2012
Age-modulated association between prefrontal NAA and the BDNF geneBasira Salehi, Nora Preuss, Jan Willem van der Veen, et al.
Annals of Neurology|August 28, 2018
A comprehensive analysis of SNCA-related genetic risk in sporadic parkinson diseaseLasse Pihlstrøm, Cornelis Blauwendraat, Chiara Cappelletti, et al.
Human Molecular Genetics|June 20, 2018
Proteomic analysis reveals co-ordinated alterations in protein synthesis and degradation pathways in LRRK2 knockout miceLaura Pellegrini, David N Hauser, Yan Li, et al.
Journal of Neurochemistry|March 19, 2005
Tyrosinase exacerbates dopamine toxicity but is not genetically associated with Parkinson's diseaseElisa Greggio, Elisabetta Bergantino, Donald Carter, et al.
Scientific Reports|February 28, 2020
In Vitro CRISPR/Cas9-Directed Gene Editing to Model LRRK2 G2019S Parkinson's Disease in Common MarmosetsScott C Vermilyea, Alexander Babinski, Nina Tran, et al.
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