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Journal of Molecular and Cellular Cardiology|March 16, 2015
Carbonic anhydrase XII in valve interstitial cells promotes the regression of calcific aortic valve stenosisRihab Bouchareb, Nancy Côté, Marie-Chloé-Boulanger, et al.
Journal of the American College of Cardiology|September 12, 2015
Oxidized Phospholipids, Lipoprotein(a), and Progression of Calcific Aortic Valve StenosisRomain Capoulade, Kwan L Chan, Calvin Yeang, et al.
Journal of Molecular and Cellular Cardiology|February 28, 2012
ATP acts as a survival signal and prevents the mineralization of aortic valveNancy Côté, Diala El Husseini, Andrée Pépin, et al.
Journal of the American Heart Association|February 15, 2018
ApoB/ApoA-I Ratio is Associated With Faster Hemodynamic Progression of Aortic Stenosis: Results From the PROGRESSA (Metabolic Determinants of the Progression of Aortic Stenosis) StudyLionel Tastet, Romain Capoulade, Mylène Shen, et al.
Cardiovascular Research|May 10, 2025
RNA interference versus antibody-based PCSK9 inhibition for the prevention of cardiovascular disease: A drug-target Mendelian randomization studyEloi Gagnon, Dipender Gill, Jérôme Bourgault, et al.
Communications Biology|November 22, 2022
Angiopoietin-like 2 is essential to aortic valve development in micePauline Labbé, Victoria Munoz Goyette, Nathalie Thorin-Trescases, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|December 17, 2013
Evaluation of links between high-density lipoprotein genetics, functionality, and aortic valve stenosis risk in humansBenoit J Arsenault, Marie-Pierre Dubé, Mathieu R Brodeur, et al.
European Heart Journal|January 20, 2024
Genetic inhibition of angiopoietin-like protein-3, lipids, and cardiometabolic riskÉmilie Gobeil, Jérôme Bourgault, Patricia L Mitchell, et al.
Circulation. Cardiovascular Genetics|November 11, 2015
Calcium Signaling Pathway Genes RUNX2 and CACNA1C Are Associated With Calcific Aortic Valve DiseaseSandra Guauque-Olarte, David Messika-Zeitoun, Arnaud Droit, et al.
Atherosclerosis|June 21, 2020
Dietary sucrose induces metabolic inflammation and atherosclerotic cardiovascular diseases more than dietary fat in LDLr<sup>-/-</sup>ApoB<sup>100/100</sup> miceLaís R Perazza, Patricia L Mitchell, Benjamin A H Jensen, et al.
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