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Science Immunology|February 10, 2023
Human IL-23 is essential for IFN-γ-dependent immunity to mycobacteriaQuentin Philippot, Masato Ogishi, Jonathan Bohlen, et al.
Annals of the Rheumatic Diseases|November 18, 2025
A de novo dominant-negative PSMB8 mutation causes severe CANDLE/PRAAS due to arrested proteasome biogenesisSophie Wolfgramm, Sara Alehashemi, Martin Wendlandt, et al.
The Journal of Experimental Medicine|June 22, 2023
Autoantibodies neutralizing type I IFNs underlie West Nile virus encephalitis in ∼40% of patientsAdrian Gervais, Francesca Rovida, Maria Antonietta Avanzini, et al.
The Journal of Experimental Medicine|March 7, 2023
Humans with inherited MyD88 and IRAK-4 deficiencies are predisposed to hypoxemic COVID-19 pneumoniaAna García-García, Rebeca Pérez de Diego, Carlos Flores, et al.
Science Translational Medicine|October 26, 2021
Identification of driver genes for critical forms of COVID-19 in a deeply phenotyped young patient cohortRaphael Carapito, Richard Li, Julie Helms, et al.
Cell|May 7, 2026
Affinity-matured B cell responses neutralizing type-I interferons underlie severe viral infectionsMorgane Fournier, Matthias Vanderkerken, Karim Dorgham, et al.
The Journal of Experimental Medicine|April 23, 2021
Preexisting autoantibodies to type I IFNs underlie critical COVID-19 pneumonia in patients with APS-1Paul Bastard, Elizaveta Orlova, Leila Sozaeva, et al.
The Journal of Experimental Medicine|April 20, 2022
A loss-of-function IFNAR1 allele in Polynesia underlies severe viral diseases in homozygotesPaul Bastard, Kuang-Chih Hsiao, Qian Zhang, et al.
The Journal of Experimental Medicine|September 16, 2022
Autoantibodies against type I IFNs in patients with critical influenza pneumoniaQian Zhang, Andrés Pizzorno, Lisa Miorin, et al.
Medrxiv : the Preprint Server for Health Sciences|April 10, 2026
High risk of hypoxemic COVID-19 pneumonia in myasthenia gravis patients with type I IFN autoantibodiesAdrian Gervais, Astrid Marchal, Alexis Maillard, et al.
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