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Neuroscience Research|June 21, 2002
Abeta-degrading endopeptidase, neprilysin, in mouse brain: synaptic and axonal localization inversely correlating with Abeta pathologyShinjiro Fukami, Kaori Watanabe, Nobuhisa Iwata, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|December 14, 2020
Reactive or transgenic increase in microglial TYROBP reveals a TREM2-independent TYROBP-APOE link in wild-type and Alzheimer's-related miceMickael Audrain, Jean-Vianney Haure-Mirande, Justyna Mleczko, et al.
Scientific Data|May 16, 2015
Metabolic differences in ripening of Solanum lycopersicum 'Ailsa Craig' and three monogenic mutantsStephan Beisken, Mark Earll, Charles Baxter, et al.
Elife|July 12, 2017
LTP and memory impairment caused by extracellular Aβ and Tau oligomers is APP-dependentDaniela Puzzo, Roberto Piacentini, Mauro Fá, et al.
Plant Biotechnology Journal|August 7, 2016
Towards the development of a sustainable soya bean-based feedstock for aquacultureHyunwoo Park, Steven Weier, Fareha Razvi, et al.
Journal of Biomedicine & Biotechnology|October 2, 2008
CD36 is significantly correlated with adipophilin in human carotid lesions and inversely correlated with plasma ApoAISophie Collot-Teixeira, Calypso Barbatis, Florence Bultelle, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 16, 2012
The β-secretase-derived C-terminal fragment of βAPP, C99, but not Aβ, is a key contributor to early intraneuronal lesions in triple-transgenic mouse hippocampusInger Lauritzen, Raphaëlle Pardossi-Piquard, Charlotte Bauer, et al.
Proteome Science|May 22, 2012
Vigilin interacts with signal peptide peptidaseStephen Hsueh-Jeng Lu, Amy Hye Won Jeon, Gerold Schmitt-Ulms, et al.
Advanced Science (Weinheim, Baden-Wurttemberg, Germany)|March 31, 2026
The Uppsala APP Mutation Promotes Wild-Type Amyloid-β Aggregation and Deposition In VivoJunyue Ge, María Pagnon de la Vega, Silvia Zampar, et al.
International Journal of Biological Macromolecules|August 20, 2025
SUMO-2 activity is inhibited by non-covalent interactions with the Aβ peptide: an exploration of potential pathogenic mechanisms in Alzheimer's diseaseValeria Ciaffaglione, Giulia Grasso, Valeria Lanza, et al.
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