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The Lancet. Neurology|February 19, 2013
Controversies and priorities in amyotrophic lateral sclerosisMartin R Turner, Orla Hardiman, Michael Benatar, et al.Annals of Neurology|May 1, 2007
Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutationsIan R A Mackenzie, Eileen H Bigio, Paul G Ince, et al.Acta Neuropathologica|March 31, 2009
Staging/typing of Lewy body related alpha-synuclein pathology: a study of the BrainNet Europe ConsortiumIrina Alafuzoff, Paul G Ince, Thomas Arzberger, et al.Acta Neuropathologica|June 21, 2007
Neuropathologic diagnostic and nosologic criteria for frontotemporal lobar degeneration: consensus of the Consortium for Frontotemporal Lobar DegenerationNigel J Cairns, Eileen H Bigio, Ian R A Mackenzie, et al.Brain : a Journal of Neurology|April 19, 2007
Large-scale pathways-based association study in amyotrophic lateral sclerosisDalia Kasperaviciute, Mike E Weale, Kevin V Shianna, et al.Journal of Neural Transmission (Vienna, Austria : 1996)|September 21, 2014
Neuropathological assessments of the pathology in frontotemporal lobar degeneration with TDP43-positive inclusions: an inter-laboratory study by the BrainNet Europe consortiumIrina Alafuzoff, Maria Pikkarainen, Manuela Neumann, et al.Acta Neuropathologica|January 21, 2014
TMEM106B is a genetic modifier of frontotemporal lobar degeneration with C9orf72 hexanucleotide repeat expansionsMichael D Gallagher, Eunran Suh, Murray Grossman, et al.Nature Genetics|February 16, 2010
Common variants at 7p21 are associated with frontotemporal lobar degeneration with TDP-43 inclusionsVivianna M Van Deerlin, Patrick M A Sleiman, Maria Martinez-Lage, et al.Pageof 10