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Plos Genetics|May 10, 2011
Reciprocal sign epistasis between frequently experimentally evolved adaptive mutations causes a rugged fitness landscapeDaniel J Kvitek, Gavin SherlockEukaryotic Cell|August 6, 2013
Ras signaling gets fine-tuned: regulation of multiple pathogenic traits of Candida albicansDiane O Inglis, Gavin SherlockPlos Biology|February 4, 2017
Seeking Goldilocks During Evolution of Drug ResistanceGavin Sherlock, Dmitri A PetrovPathology Oncology Research : POR|March 27, 2023
Corrigendum: Blood, toil, and taxoteres: Biological determinants of treatment-induced ctDNA dynamics for interpreting tumor responseChristopher T Boniface, Paul T SpellmanJournal of Biology|July 30, 2002
Evidence for large domains of similarly expressed genes in the Drosophila genomePaul T Spellman, Gerald M RubinCancer Discovery|May 16, 2012
Tackling formalin-fixed, paraffin-embedded tumor tissue with next-generation sequencingChristopher L Corless, Paul T SpellmanPathology Oncology Research : POR|June 6, 2022
Blood, toil, and taxoteres: Biological determinants of treatment-induced ctDNA dynamics for interpreting tumor responseChristopher T Boniface, Paul T SpellmanYeast (Chichester, England)|January 24, 2025
The Viral K1 Killer Yeast System: Toxicity, Immunity, and ResistanceAngelina Chan, Michelle Hays, Gavin SherlockJournal of Molecular Evolution|March 6, 2023
Fit-Seq2.0: An Improved Software for High-Throughput Fitness Measurements Using Pooled Competition AssaysFangfei Li, Jason Tarkington, Gavin SherlockBMC Genomics|May 6, 2023
An improved algorithm for inferring mutational parameters from bar-seq evolution experimentsFangfei Li, Aditya Mahadevan, Gavin SherlockPageof 23