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Mutation Research. Reviews in Mutation Research|December 4, 2013
The paradox of FGFR3 signaling in skeletal dysplasia: why chondrocytes growth arrest while other cells over proliferatePavel KrejciInvestigational New Drugs|April 27, 2007
Simple, mammalian cell-based assay for identification of inhibitors of the Erk MAP kinase pathwayPavel Krejci, Katerina Pejchalova, William R WilcoxGene|April 25, 2013
Mild clinical presentation and prolonged survival of a patient with fumarase deficiency due to the combination of a known and a novel mutation in FH geneFatih Ezgu, Pavel Krejci, Wiliam R WilcoxMolecular Genetics and Metabolism|August 8, 2007
C-natriuretic peptide: an important regulator of cartilageKaterina Pejchalova, Pavel Krejci, William R WilcoxHuman Mutation|November 3, 2011
Sixteen years and counting: the current understanding of fibroblast growth factor receptor 3 (FGFR3) signaling in skeletal dysplasiasSilvie Foldynova-Trantirkova, William R Wilcox, Pavel KrejciDevelopmental Dynamics : an Official Publication of the American Association of Anatomists|September 28, 2021
Cilia kinases in skeletal development and homeostasisSara P Abraham, Alexandru Nita, Pavel Krejci, et al.Cells|July 2, 2021
Oncogenic FGFR Fusions Produce Centrosome and Cilia Defects by Ectopic SignalingAlexandru Nita, Sara P Abraham, Pavel Krejci, et al.Orphanet Journal of Rare Diseases|June 16, 2022
A registry of achondroplasia: a 6-year experience from the Czechia and Slovak RepublicMartin Pesl, Hana Verescakova, Linda Skutkova, et al.Pediatric Research|February 23, 2007
Fibroblast growth factors 1, 2, 17, and 19 are the predominant FGF ligands expressed in human fetal growth plate cartilagePavel Krejci, Deborah Krakow, Pertchoui B Mekikian, et al.Human Mutation|July 22, 2009
Molecular pathology of the fibroblast growth factor familyPavel Krejci, Jirina Prochazkova, Vitezslav Bryja, et al.Pageof 8