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Pedro Piccardo

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Arquivos De Neuro-Psiquiatria|November 15, 2018
Complex proteinopathies and neurodegeneration: insights from the study of transmissible spongiform encephalopathiesPedro Piccardo, David M Asher
Handbook of Clinical Neurology|June 12, 2018
Dominantly inherited prion protein cerebral amyloidoses - a modern view of Gerstmann-Sträussler-ScheinkerBernardino Ghetti, Pedro Piccardo, Gianluigi Zanusso
Frontiers in Molecular Biosciences|August 30, 2017
Lysine-Directed Post-translational Modifications of Tau Protein in Alzheimer's Disease and Related TauopathiesChristiana Kontaxi, Pedro Piccardo, Andrew C Gill
Journal of the Neurological Sciences|December 17, 2017
Variable tau accumulation in murine models with abnormal prion protein depositsPedro Piccardo, Declan King, Deborah Brown, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 1, 2005
Neurodegenerative illness in transgenic mice expressing a transmembrane form of the prion proteinRichard S Stewart, Pedro Piccardo, Bernardino Ghetti, et al.
Clinics in Laboratory Medicine|May 8, 2003
Hereditary prion protein amyloidosesBernardino Ghetti, Fabrizio Tagliavini, M Takao, et al.
Clinical Anatomy (New York, N.Y.)|April 18, 2014
Human prion diseases and the risk of their transmission during anatomical dissectionBarry M Bradford, Pedro Piccardo, James W Ironside, et al.
The Journal of General Virology|April 29, 2014
Complex proteinopathy with accumulations of prion protein, hyperphosphorylated tau, α-synuclein and ubiquitin in experimental bovine spongiform encephalopathy of monkeysPedro Piccardo, Juraj Cervenak, Ming Bu, et al.
Neurobiology of Disease|July 21, 2004
GFP-tagged prion protein is correctly localized and functionally active in the brains of transgenic miceSami Barmada, Pedro Piccardo, Keiji Yamaguchi, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|December 5, 2008
Aggregated, wild-type prion protein causes neurological dysfunction and synaptic abnormalitiesRoberto Chiesa, Pedro Piccardo, Emiliano Biasini, et al.
Pageof 5

Showing results (1-10 of 49) with videos related to

Sort By:
Pageof 5
Arquivos De Neuro-Psiquiatria|November 15, 2018
Complex proteinopathies and neurodegeneration: insights from the study of transmissible spongiform encephalopathiesPedro Piccardo, David M Asher
Handbook of Clinical Neurology|June 12, 2018
Dominantly inherited prion protein cerebral amyloidoses - a modern view of Gerstmann-Sträussler-ScheinkerBernardino Ghetti, Pedro Piccardo, Gianluigi Zanusso
Frontiers in Molecular Biosciences|August 30, 2017
Lysine-Directed Post-translational Modifications of Tau Protein in Alzheimer's Disease and Related TauopathiesChristiana Kontaxi, Pedro Piccardo, Andrew C Gill
Journal of the Neurological Sciences|December 17, 2017
Variable tau accumulation in murine models with abnormal prion protein depositsPedro Piccardo, Declan King, Deborah Brown, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 1, 2005
Neurodegenerative illness in transgenic mice expressing a transmembrane form of the prion proteinRichard S Stewart, Pedro Piccardo, Bernardino Ghetti, et al.
Clinics in Laboratory Medicine|May 8, 2003
Hereditary prion protein amyloidosesBernardino Ghetti, Fabrizio Tagliavini, M Takao, et al.
Clinical Anatomy (New York, N.Y.)|April 18, 2014
Human prion diseases and the risk of their transmission during anatomical dissectionBarry M Bradford, Pedro Piccardo, James W Ironside, et al.
The Journal of General Virology|April 29, 2014
Complex proteinopathy with accumulations of prion protein, hyperphosphorylated tau, α-synuclein and ubiquitin in experimental bovine spongiform encephalopathy of monkeysPedro Piccardo, Juraj Cervenak, Ming Bu, et al.
Neurobiology of Disease|July 21, 2004
GFP-tagged prion protein is correctly localized and functionally active in the brains of transgenic miceSami Barmada, Pedro Piccardo, Keiji Yamaguchi, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|December 5, 2008
Aggregated, wild-type prion protein causes neurological dysfunction and synaptic abnormalitiesRoberto Chiesa, Pedro Piccardo, Emiliano Biasini, et al.
Pageof 5