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Peng Shang

Showing results (401-410 of 416) with videos related to

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Nature Communications|October 13, 2022
Reducing Akt2 in retinal pigment epithelial cells causes a compensatory increase in Akt1 and attenuates diabetic retinopathyHaitao Liu, Nadezda A Stepicheva, Sayan Ghosh, et al.
The Journal of Infectious Diseases|February 3, 2005
Function of HAb18G/CD147 in invasion of host cells by severe acute respiratory syndrome coronavirusZhinan Chen, Li Mi, Jing Xu, et al.
Theranostics|August 15, 2022
Therapeutic aptamer targeting sclerostin loop3 for promoting bone formation without increasing cardiovascular risk in osteogenesis imperfecta miceLuyao Wang, Yuanyuan Yu, Shuaijian Ni, et al.
Communications Biology|July 9, 2021
βA3/A1-crystallin regulates apical polarity and EGFR endocytosis in retinal pigmented epithelial cellsPeng Shang, Nadezda Stepicheva, Kenneth Teel, et al.
Biomaterials|March 31, 2015
A delivery system specifically approaching bone resorption surfaces to facilitate therapeutic modulation of microRNAs in osteoclastsJin Liu, Lei Dang, Defang Li, et al.
Communications Biology|September 26, 2019
Neutrophils homing into the retina trigger pathology in early age-related macular degenerationSayan Ghosh, Archana Padmanabhan, Tanuja Vaidya, et al.
JCI Insight|June 22, 2023
Activated cGAS/STING signaling elicits endothelial cell senescence in early diabetic retinopathyHaitao Liu, Sayan Ghosh, Tanuja Vaidya, et al.
Autophagy|April 27, 2022
Increased LCN2 (lipocalin 2) in the RPE decreases autophagy and activates inflammasome-ferroptosis processes in a mouse model of dry AMDUrvi Gupta, Sayan Ghosh, Callen T Wallace, et al.
Aging Cell|February 17, 2025
Activated mTOR Signaling in the RPE Drives EMT, Autophagy, and Metabolic Disruption, Resulting in AMD-Like Pathology in MiceOlivia Chowdhury, Sridhar Bammidi, Pooja Gautam, et al.
Nature Medicine|February 10, 2015
Aptamer-functionalized lipid nanoparticles targeting osteoblasts as a novel RNA interference-based bone anabolic strategyChao Liang, Baosheng Guo, Heng Wu, et al.
Pageof 42

Showing results (401-410 of 416) with videos related to

Sort By:
Pageof 42
Nature Communications|October 13, 2022
Reducing Akt2 in retinal pigment epithelial cells causes a compensatory increase in Akt1 and attenuates diabetic retinopathyHaitao Liu, Nadezda A Stepicheva, Sayan Ghosh, et al.
The Journal of Infectious Diseases|February 3, 2005
Function of HAb18G/CD147 in invasion of host cells by severe acute respiratory syndrome coronavirusZhinan Chen, Li Mi, Jing Xu, et al.
Theranostics|August 15, 2022
Therapeutic aptamer targeting sclerostin loop3 for promoting bone formation without increasing cardiovascular risk in osteogenesis imperfecta miceLuyao Wang, Yuanyuan Yu, Shuaijian Ni, et al.
Communications Biology|July 9, 2021
βA3/A1-crystallin regulates apical polarity and EGFR endocytosis in retinal pigmented epithelial cellsPeng Shang, Nadezda Stepicheva, Kenneth Teel, et al.
Biomaterials|March 31, 2015
A delivery system specifically approaching bone resorption surfaces to facilitate therapeutic modulation of microRNAs in osteoclastsJin Liu, Lei Dang, Defang Li, et al.
Communications Biology|September 26, 2019
Neutrophils homing into the retina trigger pathology in early age-related macular degenerationSayan Ghosh, Archana Padmanabhan, Tanuja Vaidya, et al.
JCI Insight|June 22, 2023
Activated cGAS/STING signaling elicits endothelial cell senescence in early diabetic retinopathyHaitao Liu, Sayan Ghosh, Tanuja Vaidya, et al.
Autophagy|April 27, 2022
Increased LCN2 (lipocalin 2) in the RPE decreases autophagy and activates inflammasome-ferroptosis processes in a mouse model of dry AMDUrvi Gupta, Sayan Ghosh, Callen T Wallace, et al.
Aging Cell|February 17, 2025
Activated mTOR Signaling in the RPE Drives EMT, Autophagy, and Metabolic Disruption, Resulting in AMD-Like Pathology in MiceOlivia Chowdhury, Sridhar Bammidi, Pooja Gautam, et al.
Nature Medicine|February 10, 2015
Aptamer-functionalized lipid nanoparticles targeting osteoblasts as a novel RNA interference-based bone anabolic strategyChao Liang, Baosheng Guo, Heng Wu, et al.
Pageof 42