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Peter A Bell

Showing results (1-10 of 27) with videos related to

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International Journal of Molecular Sciences|May 27, 2023
No Substrate Left behind-Mining of Shotgun Proteomics Datasets Rescues Evidence of Proteolysis by SARS-CoV-2 3CL<sup>pro</sup> Main ProteasePeter A Bell, Christopher M Overall
Mayo Clinic Proceedings|February 5, 2008
What do we really know about neurological misdiagnosis in the emergency department?Janice Wachtler, Peter A Bell, Thomas Brabson
International Journal of Molecular Medicine|April 1, 2015
The utility of mouse models to provide information regarding the pathomolecular mechanisms in human genetic skeletal diseases: The emerging role of endoplasmic reticulum stress (Review)Michael D Briggs, Peter A Bell, Katarzyna A Pirog
European Journal of Human Genetics : EJHG|March 6, 2014
Genotype to phenotype correlations in cartilage oligomeric matrix protein associated chondrodysplasiasMichael D Briggs, Joanne Brock, Simon C Ramsden, et al.
Analytical Biochemistry|May 26, 2004
A high-affinity reversible protein stain for Western blotsBabu S Antharavally, Brad Carter, Peter A Bell, et al.
Expert Opinion on Orphan Drugs|December 5, 2015
New therapeutic targets in rare genetic skeletal diseasesMichael D Briggs, Peter A Bell, Michael J Wright, et al.
Journal of Proteome Research|October 12, 2019
Proteomic and N-Terminomic TAILS Analyses of Human Alveolar Bone Proteins: Improved Protein Extraction Methodology and LysargiNase Digestion Strategies Increase Proteome Coverage and Missing Protein IdentificationPeter A Bell, Nestor Solis, Jayachandran N Kizhakkedathu, et al.
Analytical Biochemistry|December 17, 2008
Quantitation of proteins using a dye-metal-based colorimetric protein assayBabu S Antharavally, Krishna A Mallia, Priya Rangaraj, et al.
Arthritis and Rheumatism|November 16, 2011
Loss of matrilin 1 does not exacerbate the skeletal phenotype in a mouse model of multiple epiphyseal dysplasia caused by a Matn3 V194D mutationPeter A Bell, Katarzyna A Piróg, Maryline Fresquet, et al.
Disease Models & Mechanisms|September 19, 2013
A novel transgenic mouse model of growth plate dysplasia reveals that decreased chondrocyte proliferation due to chronic ER stress is a key factor in reduced bone growthBenedetta Gualeni, M Helen Rajpar, Aaron Kellogg, et al.
Pageof 3

Showing results (1-10 of 27) with videos related to

Sort By:
Pageof 3
International Journal of Molecular Sciences|May 27, 2023
No Substrate Left behind-Mining of Shotgun Proteomics Datasets Rescues Evidence of Proteolysis by SARS-CoV-2 3CL<sup>pro</sup> Main ProteasePeter A Bell, Christopher M Overall
Mayo Clinic Proceedings|February 5, 2008
What do we really know about neurological misdiagnosis in the emergency department?Janice Wachtler, Peter A Bell, Thomas Brabson
International Journal of Molecular Medicine|April 1, 2015
The utility of mouse models to provide information regarding the pathomolecular mechanisms in human genetic skeletal diseases: The emerging role of endoplasmic reticulum stress (Review)Michael D Briggs, Peter A Bell, Katarzyna A Pirog
European Journal of Human Genetics : EJHG|March 6, 2014
Genotype to phenotype correlations in cartilage oligomeric matrix protein associated chondrodysplasiasMichael D Briggs, Joanne Brock, Simon C Ramsden, et al.
Analytical Biochemistry|May 26, 2004
A high-affinity reversible protein stain for Western blotsBabu S Antharavally, Brad Carter, Peter A Bell, et al.
Expert Opinion on Orphan Drugs|December 5, 2015
New therapeutic targets in rare genetic skeletal diseasesMichael D Briggs, Peter A Bell, Michael J Wright, et al.
Journal of Proteome Research|October 12, 2019
Proteomic and N-Terminomic TAILS Analyses of Human Alveolar Bone Proteins: Improved Protein Extraction Methodology and LysargiNase Digestion Strategies Increase Proteome Coverage and Missing Protein IdentificationPeter A Bell, Nestor Solis, Jayachandran N Kizhakkedathu, et al.
Analytical Biochemistry|December 17, 2008
Quantitation of proteins using a dye-metal-based colorimetric protein assayBabu S Antharavally, Krishna A Mallia, Priya Rangaraj, et al.
Arthritis and Rheumatism|November 16, 2011
Loss of matrilin 1 does not exacerbate the skeletal phenotype in a mouse model of multiple epiphyseal dysplasia caused by a Matn3 V194D mutationPeter A Bell, Katarzyna A Piróg, Maryline Fresquet, et al.
Disease Models & Mechanisms|September 19, 2013
A novel transgenic mouse model of growth plate dysplasia reveals that decreased chondrocyte proliferation due to chronic ER stress is a key factor in reduced bone growthBenedetta Gualeni, M Helen Rajpar, Aaron Kellogg, et al.
Pageof 3