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Cardiovascular Toxicology|February 23, 2013
FGF-2 and FGF-16 protect isolated perfused mouse hearts from acute doxorubicin-induced contractile dysfunctionDavid P Sontag, Jie Wang, Elissavet Kardami, et al.
Cardiovascular Research|December 31, 2002
Biological activities of fibroblast growth factor-2 in the adult myocardiumKaren A Detillieux, Farah Sheikh, Elissavet Kardami, et al.
Endocrine Connections|October 28, 2020
Sleep deprivation and diet affect human GH gene expression in transgenic mice in vivoJessica S Jarmasz, Yan Jin, Hana Vakili, et al.
Cardiovascular Research|January 24, 2013
FGF-2 protects cardiomyocytes from doxorubicin damage via protein kinase C-dependent effects on efflux transportersJie Wang, Mark W Nachtigal, Elissavet Kardami, et al.
The Journal of Physiological Sciences : JPS|April 14, 2011
A single bout of exercise promotes sustained left ventricular function improvement after isoproterenol-induced injury in miceSarah K Jimenez, Davinder S Jassal, Elissavet Kardami, et al.
Molecular and Cellular Biochemistry|May 11, 2011
Protection by endogenous FGF-2 against isoproterenol-induced cardiac dysfunction is attenuated by cyclosporine ASarah K Jimenez, Davinder S Jassal, Elissavet Kardami, et al.
Molecular and Cellular Biochemistry|July 5, 2003
High levels of CUG-initiated FGF-2 expression cause chromatin compaction, decreased cardiomyocyte mitosis, and cell deathCheryl J A Hirst, Meenhard Herlyn, Peter A Cattini, et al.
DNA and Cell Biology|September 20, 2018
Cardiac Fgf-16 Expression Supports Cardiomyocyte Survival and Increases Resistance to Doxorubicin CytotoxicityJie Wang, Bo Xiang, Vernon W Dolinsky, et al.
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