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Antioxidants (Basel, Switzerland)|February 23, 2018
Nanotherapy and Reactive Oxygen Species (ROS) in Cancer: A Novel PerspectivePeter Brenneisen, Andreas S ReichertMolecular Aspects of Medicine|August 18, 2005
Selenium, oxidative stress, and health aspectsPeter Brenneisen, Holger Steinbrenner, Helmut SiesAnnals of the New York Academy of Sciences|December 18, 2002
Ultraviolet-B irradiation and matrix metalloproteinases: from induction via signaling to initial eventsPeter Brenneisen, Helmut Sies, Karin Scharffetter-KochanekBioscience Reports|December 6, 2016
Fibroblast-to-myofibroblast switch is mediated by NAD(P)H oxidase generated reactive oxygen speciesLirija Alili, Maren Sack, Katharina Puschmann, et al.Toxicology Letters|July 22, 2018
The BH3 mimetic compound BH3I-1 impairs mitochondrial dynamics and promotes stress response in addition to its pro-apoptotic key functionDavid Stucki, Peter Brenneisen, Andreas S Reichert, et al.Biological Chemistry|October 17, 2007
Post-translational processing of selenoprotein P: implications of glycosylation for its utilisation by target cellsHolger Steinbrenner, Lirija Alili, Dominik Stuhlmann, et al.Free Radical Biology & Medicine|April 25, 2006
Involvement of selenoprotein P in protection of human astrocytes from oxidative damageHolger Steinbrenner, Lirija Alili, Esra Bilgic, et al.Free Radical Research|October 4, 2006
Selenoprotein P protects endothelial cells from oxidative damage by stimulation of glutathione peroxidase expression and activityHolger Steinbrenner, Esra Bilgic, Lirija Alili, et al.Redox Biology|December 6, 2014
Redox-active cerium oxide nanoparticles protect human dermal fibroblasts from PQ-induced damageClaudia von Montfort, Lirija Alili, Sarah Teuber-Hanselmann, et al.Age (Dordrecht, Netherlands)|May 17, 2014
A drug-induced accelerated senescence (DIAS) is a possibility to study aging in time lapseLirija Alili, Johanna Diekmann, Melanie Giesen, et al.Pageof 5