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Peter G Nikkels

Showing results (1-10 of 5) with videos related to

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Stroke|January 14, 2016
Cortical Sparing in Preterm Ischemic Arterial StrokeNiek E van der Aa, Manon J N L Benders, Peter G Nikkels, et al.
American Journal of Medical Genetics. Part A|September 5, 2003
Prenatal diagnosis of boomerang dysplasiaMarja W Wessels, Nicolette S Den Hollander, Ronald R De Krijger, et al.
Pediatric Radiology|July 3, 2017
Apparent diffusion coefficient as it relates to histopathology findings in post-chemotherapy nephroblastoma: a feasibility studyAnnemieke S Littooij, Peter G Nikkels, Christina A Hulsbergen-van de Kaa, et al.
Nature Medicine|October 24, 2007
Nucleotide-sugar transporter SLC35D1 is critical to chondroitin sulfate synthesis in cartilage and skeletal development in mouse and humanShuichi Hiraoka, Tatsuya Furuichi, Gen Nishimura, et al.
American Journal of Human Genetics|October 25, 2011
Craniosynostosis and multiple skeletal anomalies in humans and zebrafish result from a defect in the localized degradation of retinoic acidKathrin Laue, Hans-Martin Pogoda, Philip B Daniel, et al.
Pageof 1

Showing results (1-10 of 5) with videos related to

Sort By:
Pageof 1
Stroke|January 14, 2016
Cortical Sparing in Preterm Ischemic Arterial StrokeNiek E van der Aa, Manon J N L Benders, Peter G Nikkels, et al.
American Journal of Medical Genetics. Part A|September 5, 2003
Prenatal diagnosis of boomerang dysplasiaMarja W Wessels, Nicolette S Den Hollander, Ronald R De Krijger, et al.
Pediatric Radiology|July 3, 2017
Apparent diffusion coefficient as it relates to histopathology findings in post-chemotherapy nephroblastoma: a feasibility studyAnnemieke S Littooij, Peter G Nikkels, Christina A Hulsbergen-van de Kaa, et al.
Nature Medicine|October 24, 2007
Nucleotide-sugar transporter SLC35D1 is critical to chondroitin sulfate synthesis in cartilage and skeletal development in mouse and humanShuichi Hiraoka, Tatsuya Furuichi, Gen Nishimura, et al.
American Journal of Human Genetics|October 25, 2011
Craniosynostosis and multiple skeletal anomalies in humans and zebrafish result from a defect in the localized degradation of retinoic acidKathrin Laue, Hans-Martin Pogoda, Philip B Daniel, et al.
Pageof 1