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Journal of Diabetes Science and Technology|January 22, 2019
Model-Based Tool for Personalized Adjustment of Basal Insulin Supply in Patients With Intensified Conventional Insulin TherapyLutz Vogt, Andreas Thomas, Gert Fritzsche, et al.Herz|September 2, 2004
[The metabolic syndrome in patients with type 1 diabetes mellitus. Associations with cardiovascular risk factors and cardiovascular morbidity]Jörg Reindel, Eckhard Zander, Peter Heinke, et al.Diabetic Medicine : a Journal of the British Diabetic Association|July 13, 2025
Residual insulin secretion in long-standing type 1 diabetesPetra Augstein, Natalie Buschmann, Janik Riese, et al.Biochemical and Biophysical Research Communications|April 25, 2003
Prevention of autoimmune diabetes in NOD mice by troglitazone is associated with modulation of ICAM-1 expression on pancreatic islet cells and IFN-gamma expression in splenic T cellsPetra Augstein, Annemarie Dunger, Peter Heinke, et al.Journal of Autoimmunity|June 7, 2003
IL-1beta, IFN-gamma and TNF-alpha increase vulnerability of pancreatic beta cells to autoimmune destructionGerhild Wachlin, Petra Augstein, Dieter Schröder, et al.Diabetes Care|May 1, 2007
Outpatient assessment of Karlsburg Diabetes Management System-based decision supportPetra Augstein, Lutz Vogt, Klaus-Dieter Kohnert, et al.BMC Endocrine Disorders|May 2, 2015
Q-Score: development of a new metric for continuous glucose monitoring that enables stratification of antihyperglycaemic therapiesPetra Augstein, Peter Heinke, Lutz Vogt, et al.Journal of Autoimmunity|December 2, 2004
Cytokines activate caspase-3 in insulinoma cells of diabetes-prone NOD mice directly and via upregulation of FasPetra Augstein, Jeanette Bahr, Gerhild Wachlin, et al.Diabetes & Vascular Disease Research|September 9, 2008
Dominance of cytokine- over FasL-induced impairment of the mitochondrial transmembrane potential (Deltapsim) in the pancreatic beta-cell line NIT-1Petra Augstein, Peter Heinke, Eckhard Salzsieder, et al.Diabetes Research and Clinical Practice|March 3, 2007
Chronic hyperglycemia but not glucose variability determines HbA1c levels in well-controlled patients with type 2 diabetesKlaus-Dieter Kohnert, Petra Augstein, Peter Heinke, et al.Pageof 3