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Current Opinion in Cardiology|April 30, 2005
Ankyrin-based cardiac arrhythmias: a new class of channelopathies due to loss of cellular targetingPeter J Mohler, Vann Bennett
Frontiers in Bioscience : a Journal and Virtual Library|June 23, 2005
Defects in ankyrin-based cellular pathways in metazoan physiologyPeter J Mohler, Vann Bennett
The Journal of Biological Chemistry|July 21, 2004
Ankyrin-B targets beta2-spectrin to an intracellular compartment in neonatal cardiomyocytesPeter J Mohler, Woohyun Yoon, Vann Bennett
The Journal of Biological Chemistry|January 10, 2002
The ankyrin-B C-terminal domain determines activity of ankyrin-B/G chimeras in rescue of abnormal inositol 1,4,5-trisphosphate and ryanodine receptor distribution in ankyrin-B (-/-) neonatal cardiomyocytesPeter J Mohler, Anthony O Gramolini, Vann Bennett
Plos Biology|November 19, 2005
Ankyrin-B coordinates the Na/K ATPase, Na/Ca exchanger, and InsP3 receptor in a cardiac T-tubule/SR microdomainPeter J Mohler, Jonathan Q Davis, Vann Bennett
The Journal of Biological Chemistry|December 22, 2005
Isoform specificity of ankyrin-B: a site in the divergent C-terminal domain is required for intramolecular associationKhadar M Abdi, Peter J Mohler, Jonathan Q Davis, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 19, 2019
βII-spectrin promotes mouse brain connectivity through stabilizing axonal plasma membranes and enabling axonal organelle transportDamaris N Lorenzo, Alexandra Badea, Ruobo Zhou, et al.
The Journal of Biological Chemistry|November 1, 2006
Ankyrin-G and beta2-spectrin collaborate in biogenesis of lateral membrane of human bronchial epithelial cellsKrishnakumar Kizhatil, Woohyun Yoon, Peter J Mohler, et al.
The Journal of Biological Chemistry|January 15, 2004
Inositol 1,4,5-trisphosphate receptor localization and stability in neonatal cardiomyocytes requires interaction with ankyrin-BPeter J Mohler, Jonathan Q Davis, Lydia H Davis, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 8, 2004
Nav1.5 E1053K mutation causing Brugada syndrome blocks binding to ankyrin-G and expression of Nav1.5 on the surface of cardiomyocytesPeter J Mohler, Ilaria Rivolta, Carlo Napolitano, et al.
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