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Clinical Journal of the American Society of Nephrology : CJASN|May 31, 2014
Collecting duct principal cell transport processes and their regulationDavid Pearce, Rama Soundararajan, Christiane Trimpert, et al.
The Journal of Biological Chemistry|February 18, 2011
Hypotonicity-induced reduction of aquaporin-2 transcription in mpkCCD cells is independent of the tonicity responsive element, vasopressin, and cAMPMarleen L A Kortenoeven, Michiel van den Brand, Jack F M Wetzels, et al.
Nephrology, Dialysis, Transplantation : Official Publication of the European Dialysis and Transplant Association - European Renal Association|July 11, 2012
Vasopressin increases S261 phosphorylation in AQP2-P262L, a mutant in recessive nephrogenic diabetes insipidusChristiane Trimpert, Dennis T M van den Berg, Robert A Fenton, et al.
Seminars in Nephrology|June 4, 2008
Aquaporin 2 mutations in nephrogenic diabetes insipidusAnne J M Loonen, Nine V A M Knoers, Carel H van Os, et al.
American Journal of Physiology. Renal Physiology|May 23, 2008
Renal expression of exchange protein directly activated by cAMP (Epac) 1 and 2Yuedan Li, Irene B M Konings, Jun Zhao, et al.
American Journal of Physiology. Renal Physiology|April 16, 2010
RasGRP1 stimulation enhances ubiquitination and endocytosis of the sodium-chloride cotransporterBenjamin Ko, Erik-Jan Kamsteeg, Leslie L Cooke, et al.
Physiological Genomics|August 11, 2021
Genetic background determines renal response to chronic lithium treatment in female miceTheun de Groot, Rosalinda Doty, Lars Damen, et al.
Journal of the American Society of Nephrology : JASN|January 11, 2014
Lithium causes G2 arrest of renal principal cellsTheun de Groot, Mohammad Alsady, Marcel Jaklofsky, et al.
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