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FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|September 12, 2012
CO2 permeability of cell membranes is regulated by membrane cholesterol and protein gas channelsFabian Itel, Samer Al-Samir, Fredrik Öberg, et al.Journal of the American Society of Nephrology : JASN|August 27, 2005
Lack of arginine vasopressin-induced phosphorylation of aquaporin-2 mutant AQP2-R254L explains dominant nephrogenic diabetes insipidusFabrizio de Mattia, Paul J M Savelkoul, Erik-Jan Kamsteeg, et al.Journal of the American Society of Nephrology : JASN|April 5, 2014
A protein kinase A-independent pathway controlling aquaporin 2 trafficking as a possible cause for the syndrome of inappropriate antidiuresis associated with polycystic kidney disease 1 haploinsufficiencyGrazia Tamma, Domenica Lasorsa, Christiane Trimpert, et al.Journal of the American Society of Nephrology : JASN|April 22, 2004
Characterization of an aquaporin-2 water channel gene mutation causing partial nephrogenic diabetes insipidus in a Mexican family: evidence of increased frequency of the mutation in the town of originCristina Boccalandro, Fabrizio De Mattia, Dong-Chuan Guo, et al.Human Molecular Genetics|April 4, 2002
Heteroligomerization of an Aquaporin-2 mutant with wild-type Aquaporin-2 and their misrouting to late endosomes/lysosomes explains dominant nephrogenic diabetes insipidusNannette Marr, Daniel G Bichet, Michele Lonergan, et al.Proceedings of the National Academy of Sciences of the United States of America|July 10, 2009
Intracellular activation of vasopressin V2 receptor mutants in nephrogenic diabetes insipidus by nonpeptide agonistsJoris H Robben, Marleen L A Kortenoeven, Mozes Sze, et al.The Journal of Biological Chemistry|March 23, 2006
Quaternary ammonium compounds as water channel blockers. Specificity, potency, and site of actionFrank J M Detmers, Bert L de Groot, E Matthias Müller, et al.Pflugers Archiv : European Journal of Physiology|October 30, 2007
Missorting of the Aquaporin-2 mutant E258K to multivesicular bodies/lysosomes in dominant NDI is associated with its monoubiquitination and increased phosphorylation by PKC but is due to the loss of E258Erik-Jan Kamsteeg, Paul J M Savelkoul, Giel Hendriks, et al.American Journal of Physiology. Renal Physiology|October 27, 2017
Lithium induces aerobic glycolysis and glutaminolysis in collecting duct principal cellsMohammad Alsady, Theun de Groot, Marleen L A Kortenoeven, et al.Biochimica Et Biophysica Acta|April 25, 2006
Polarisation, key to good localisationMoniek van Beest, Joris H Robben, Paul J M Savelkoul, et al.Pageof 9