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Peter Proks

Showing results (31-40 of 55) with videos related to

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Proceedings of the National Academy of Sciences of the United States of America|December 8, 2004
Molecular basis of Kir6.2 mutations associated with neonatal diabetes or neonatal diabetes plus neurological featuresPeter Proks, Jennifer F Antcliff, Jon Lippiat, et al.
Wellcome Open Research|August 10, 2021
Phenotype of a transient neonatal diabetes point mutation (SUR1-R1183W) in miceGregor Sachse, Elizabeth Haythorne, Peter Proks, et al.
Pflugers Archiv : European Journal of Physiology|October 6, 2006
Functional analysis of six Kir6.2 (KCNJ11) mutations causing neonatal diabetesChristophe A J Girard, Kenju Shimomura, Peter Proks, et al.
Diabetologia|April 28, 2016
Neonatal diabetes caused by a homozygous KCNJ11 mutation demonstrates that tiny changes in ATP sensitivity markedly affect diabetes riskNatascia Vedovato, Edward Cliff, Peter Proks, et al.
Endocrine Journal|September 8, 2009
A proposal of combined evaluation of waist circumference and BMI for the diagnosis of metabolic syndromeMasaki Takahashi, Kenju Shimomura, Peter Proks, et al.
EMBO Reports|May 3, 2005
A gating mutation at the internal mouth of the Kir6.2 pore is associated with DEND syndromePeter Proks, Christophe Girard, Shozeb Haider, et al.
Journal of Food and Drug Analysis|June 1, 2022
Identification of oxytocin receptor activating chemical components from traditional Japanese medicinesYuko Maejima, Shoichiro Horita, Shoko Yokota, et al.
The EMBO Journal|February 25, 2026
Structural determinants for GPCR-mediated inhibition of TASK K2P channels by diacylglycerol and its dysfunction in diseaseThibault R H Jouen-Tachoire, Peter Proks, David Seiferth, et al.
The Journal of General Physiology|May 25, 2021
Norfluoxetine inhibits TREK-2 K2P channels by multiple mechanisms including state-independent effects on the selectivity filter gatePeter Proks, Marcus Schewe, Linus J Conrad, et al.
Human Molecular Genetics|April 15, 2006
A heterozygous activating mutation in the sulphonylurea receptor SUR1 (ABCC8) causes neonatal diabetesPeter Proks, Amanda L Arnold, Jan Bruining, et al.
Pageof 6

Showing results (31-40 of 55) with videos related to

Sort By:
Pageof 6
Proceedings of the National Academy of Sciences of the United States of America|December 8, 2004
Molecular basis of Kir6.2 mutations associated with neonatal diabetes or neonatal diabetes plus neurological featuresPeter Proks, Jennifer F Antcliff, Jon Lippiat, et al.
Wellcome Open Research|August 10, 2021
Phenotype of a transient neonatal diabetes point mutation (SUR1-R1183W) in miceGregor Sachse, Elizabeth Haythorne, Peter Proks, et al.
Pflugers Archiv : European Journal of Physiology|October 6, 2006
Functional analysis of six Kir6.2 (KCNJ11) mutations causing neonatal diabetesChristophe A J Girard, Kenju Shimomura, Peter Proks, et al.
Diabetologia|April 28, 2016
Neonatal diabetes caused by a homozygous KCNJ11 mutation demonstrates that tiny changes in ATP sensitivity markedly affect diabetes riskNatascia Vedovato, Edward Cliff, Peter Proks, et al.
Endocrine Journal|September 8, 2009
A proposal of combined evaluation of waist circumference and BMI for the diagnosis of metabolic syndromeMasaki Takahashi, Kenju Shimomura, Peter Proks, et al.
EMBO Reports|May 3, 2005
A gating mutation at the internal mouth of the Kir6.2 pore is associated with DEND syndromePeter Proks, Christophe Girard, Shozeb Haider, et al.
Journal of Food and Drug Analysis|June 1, 2022
Identification of oxytocin receptor activating chemical components from traditional Japanese medicinesYuko Maejima, Shoichiro Horita, Shoko Yokota, et al.
The EMBO Journal|February 25, 2026
Structural determinants for GPCR-mediated inhibition of TASK K2P channels by diacylglycerol and its dysfunction in diseaseThibault R H Jouen-Tachoire, Peter Proks, David Seiferth, et al.
The Journal of General Physiology|May 25, 2021
Norfluoxetine inhibits TREK-2 K2P channels by multiple mechanisms including state-independent effects on the selectivity filter gatePeter Proks, Marcus Schewe, Linus J Conrad, et al.
Human Molecular Genetics|April 15, 2006
A heterozygous activating mutation in the sulphonylurea receptor SUR1 (ABCC8) causes neonatal diabetesPeter Proks, Amanda L Arnold, Jan Bruining, et al.
Pageof 6